Doctoremia
Kanalga Telegramâda oâtish
A doctor remains a student until his last breath. The moment he stops learning, he truly begins to die
Ko'proq ko'rsatish6 496
Obunachilar
Ma'lumot yo'q24 soatlar
+47 kunlar
+7930 kunlar
Postlar arxiv
6 496
Massive PE + shock đ¨
=Immediate systemic thrombolysis (Alteplase) đ
Massive PE + contraindication to thrombolysis đ
=Surgical embolectomy or catheter-directed thrombectomy đĨ
Massive PE + failed thrombolysis â
=Surgical embolectomy or catheter intervention đ ī¸
Stable PE â
=Anticoagulation only (DOAC/LMWH đ)
Cardiac arrest from PE đ
=CPR + thrombolysis đ
6 496
+2
Patient presents with palpitations
ECG :
Short PR interval
Upsloping QRS :- Delta waves
Dx: Wolff-Parkinson-White (WPW) syndrome
This is type B as S>R in V1
6 496
Sometimes, despite our sincerest intentions and our greatest efforts, a patient may still slip beyond our reach. Such is the weight we carry in this profession. Healing is our duty, but life and death remain in the hands of Allah.
Keep your faith firm. Continue to seek knowledge with humility, for every lesson makes us better servants to humanity. May Allah increase us all in beneficial knowledge, wisdom, and compassion.
Medicine is a profession that reaches far beyond money or prestige. It is a sacred trust. It is about protecting human life, easing suffering, and serving Allahâs most honored creation with sincerity and mercy.
Every soul is destined to taste death. Its appointed time is already written, and no one can hasten it or delay it by even a single moment. Our responsibility is not to control destiny, but to strive with excellence, kindness, and integrity until our very last breath.
May Allah accept our efforts, forgive our shortcomings, and grant healing where healing is written, patience where trials remain, and peace to every soul when its journey returns to Him.
6 496
Davidson 25th
đā§§.SLE + Antiphospholipid antibodies (+), āĻāĻŋāύā§āϤ⧠āĻāĻā§ āĻāĻāύāĻ thrombosis āĻšāϝāĻŧāύāĻŋ
âPatients with SLE positive for antiphospholipid antibodies, without previous episodes of thrombosis, should be given prophylaxis with low-dose aspirin.â
āĻāϰ āĻŽāĻžāύ⧠āĻā§?
SLE āϰā§āĻā§āĻĻā§āϰ āĻŽāϧā§āϝ⧠āĻ
āύā§āĻā§āϰ āĻļāϰā§āϰ⧠Antiphospholipid antibodies (aPL) āĻĨāĻžāĻā§āĨ¤
āĻāĻā§āϞ⧠āĻšāϞā§:
* Lupus anticoagulant
* Anticardiolipin antibody
* Anti-β2 glycoprotein I antibody
āύāĻžāĻŽ anticoagulant āĻšāϞā§āĻ āĻļāϰā§āϰā§āϰ āĻā§āϤāϰ⧠āĻāĻā§āϞ⧠clot āϤā§āϰāĻŋ āĻāϰāĻžāϰ āĻĒā§āϰāĻŦāĻŖāϤāĻž āĻŦāĻžāĻĄāĻŧāĻžāϝāĻŧāĨ¤
āϤāĻžāĻ āϝāĻĻāĻŋ:
* SLE āĻĨāĻžāĻā§
* aPL positive āĻĨāĻžāĻā§
* āĻāĻŋāύā§āϤ⧠āĻāĻāύāĻ āĻā§āύ⧠clot (DVT, Stroke, PE āĻāϤā§āϝāĻžāĻĻāĻŋ) āύāĻž āĻšāϝāĻŧā§ āĻĨāĻžāĻā§
āϤāĻžāĻšāϞ⧠āĻāĻŦāĻŋāώā§āϝāϤ⧠clot āĻšāĻāϝāĻŧāĻž āĻ ā§āĻāĻžāϤ⧠Low-dose Aspirin (75â100 mg/day) āĻĻā§āĻāϝāĻŧāĻž āĻšāϝāĻŧāĨ¤
âĄī¸ āĻāĻāĻžāĻā§ āĻŦāϞ⧠Primary prophylaxisāĨ¤
⧍. āϝāĻĻāĻŋ āĻāĻā§ thrombosis āĻšāϝāĻŧā§ āĻĨāĻžāĻā§
âThose with previous episodes of thrombosis require lifelong anticoagulant therapy.â
āϝāĻĻāĻŋ āĻāϤā§āĻŽāϧā§āϝ⧠āϰā§āĻā§āϰ:
* DVT
* Pulmonary embolism
* Stroke
* MI
* Arterial thrombosis
āĻšāϝāĻŧā§ āĻĨāĻžāĻā§,
āϤāĻžāĻšāϞ⧠aspirin āϝāĻĨā§āώā§āĻ āύāϝāĻŧāĨ¤
āĻĻāĻŋāϤ⧠āĻšāĻŦā§ Life-long anticoagulationāĨ¤
āϝā§āĻŽāύ:
* Warfarin
* (āĻāĻŋāĻā§ āĻā§āώā§āϤā§āϰā§) Apixaban āĻŦāĻž āĻ
āύā§āϝ DOAC
āĻā§āύ?
āĻāĻžāϰāĻŖ āĻāĻāĻŦāĻžāϰ clot āĻšāϞ⧠āĻāĻŦāĻžāϰ clot āĻšāĻāϝāĻŧāĻžāϰ āĻā§āĻāĻāĻŋ āĻ
āύā§āĻ āĻŦā§āĻļāĻŋāĨ¤
āĻāĻāĻŋ āĻšāϞ⧠Secondary prophylaxisāĨ¤
đŧāĻŽāύ⧠āϰāĻžāĻāĻžāϰ āϏāĻšāĻ āĻā§āϰāĻŋāĻ
No clot â Aspirin
Previous clot â Lifelong anticoagulant
ā§Š. SLE āϰā§āĻā§āĻĻā§āϰ Osteoporosis-āĻāϰ āĻā§āĻāĻāĻŋ āĻā§āύ āĻŦā§āĻļāĻŋ?
âPatients with SLE are at risk of osteoporosis.â
āĻāĻžāϰāĻŖ āĻ
āύā§āĻāĻā§āϞ⧠:
(āĻ) āĻĻā§āϰā§āĻāĻĻāĻŋāύ Steroid āĻŦā§āϝāĻŦāĻšāĻžāϰ
Prednisolone:
* Osteoblast activity āĻāĻŽāĻžāϝāĻŧ
* Osteoclast activity āĻŦāĻžāĻĄāĻŧāĻžāϝāĻŧ
* Calcium absorption āĻāĻŽāĻžāϝāĻŧ
* Bone resorption āĻŦāĻžāĻĄāĻŧāĻžāϝāĻŧ
āĻĢāϞ⧠Bone mineral density āĻāĻŽā§ āϝāĻžāϝāĻŧāĨ¤
(āĻ) Chronic inflammation
SLE-āĻāϰ cytokine-mediated inflammation āύāĻŋāĻā§āĻ bone loss āĻāĻāĻžāϝāĻŧāĨ¤
(āĻ) Vitamin D deficiency
SLE āϰā§āĻā§āϰāĻž āĻ
āύā§āĻ āϏāĻŽāϝāĻŧ āϏā§āϰā§āϝā§āϰ āĻāϞ⧠āĻāĻĄāĻŧāĻŋāϝāĻŧā§ āĻāϞā§āύ (photosensitivity-āĻāϰ āĻāĻžāϰāĻŖā§), āĻĢāϞ⧠Vitamin D āĻāĻŽā§ āϝāĻžāϝāĻŧāĨ¤
ā§Ē. DXA Scan āĻā§āύ āĻāϰāĻž āĻšāϝāĻŧ?
âDXA should be consideredâĻâ
DXA = Dual-energy X-ray Absorptiometry
āĻāĻāĻŋ Bone Mineral Density (BMD) āĻŽāĻžāĻĒā§āĨ¤
āĻ
āϰā§āĻĨāĻžā§ osteoporosis āĻšāϝāĻŧā§āĻā§ āĻāĻŋ āύāĻž āϤāĻž āύāĻŋāϰā§āĻŖāϝāĻŧ āĻāϰā§āĨ¤
āĻāĻāύ DXA āĻāϰāϤ⧠āĻšāĻŦā§?
(ā§§)
āϝāĻĻāĻŋ Prednisolone:
>7.5 mg/day
āĻāĻŦāĻ
ā§Š āĻŽāĻžāϏā§āϰ āĻŦā§āĻļāĻŋ
āĻāϞāϤ⧠āĻĨāĻžāĻā§āĨ¤
āĻāĻžāϰāĻŖ āĻāϤāĻĻāĻŋāύ steroid āύāĻŋāϞ⧠osteoporosis-āĻāϰ āĻā§āĻāĻāĻŋ āĻāϞā§āϞā§āĻāϝā§āĻā§āϝāĻāĻžāĻŦā§ āĻŦā§āĻĄāĻŧā§ āϝāĻžāϝāĻŧāĨ¤
(⧍)
āϝāĻĻāĻŋ āϰā§āĻā§āϰ
10-year fracture risk >10%
āĻšāϝāĻŧāĨ¤
āĻāĻāĻŋ āϏāĻžāϧāĻžāϰāĻŖāϤ FRAX score āĻĻāĻŋāϝāĻŧā§ āĻšāĻŋāϏāĻžāĻŦ āĻāϰāĻž āĻšāϝāĻŧāĨ¤
āĻ
āϰā§āĻĨāĻžā§ āĻāĻāĻžāĻŽā§ ā§§ā§Ļ āĻŦāĻāϰ⧠āĻšāĻžāĻĄāĻŧ āĻāĻžāĻāĻžāϰ āϏāĻŽā§āĻāĻžāĻŦāύāĻž ā§§ā§Ļ%-āĻāϰ āĻŦā§āĻļāĻŋ āĻšāϞ⧠DXA āĻāϰāĻž āĻāĻāĻŋāϤāĨ¤
āĻĄāĻž āϤāĻžāĻŽāĻŋāĻŽ
6 496
A 20 years old male comes to you with multiple episodes of hemoptysis and passage of bulky pale stool for the last 5 days. He has been recently diagnosed with DM and takes insulin. His genotype testing reveals a gene defect on a long arm of chromosome. Following of which second messenger system controls this gene?
A. cGMP
B. cAMP
C. Calcium phospholipid system
D. Tyrosine kinase
E. None of the above
6 496
+1
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6 496
A 70-year-old male patient with End-Stage Renal Disease (ESRD) undergoing hemodialysis (MHD) via a permanent HD catheter presents with fever and shivering đĄī¸đ¤. Which pathogen is the most common causative agent in this clinical scenario? đĻ đ
6 496
+1
āĻāĻĒāύāĻžāĻĻā§āϰ āϏāĻŦāĻžāϰ āĻŦāĻŋāĻļā§āĻŦāϏā§āϤ āĻĒā§āϰāϤāĻŋāώā§āĻ āĻžāύ Mediverse āĻŦāĻŋāϏāĻŋāĻāϏ āύāĻŋā§ā§ āĻĒāĻžāĻŦāϞāĻŋāĻļ āĻāϰāϤ⧠āϝāĻžāĻā§āĻā§ āϏā§āϰāĻž āĻĻā§āĻā§ āĻŦāĻ!!āĻ
āϰā§āĻĄāĻžāϰ āĻāϰāϤ⧠āĻāĻŽā§āύā§āĻ āĻāϰā§āύāĨ¤
6 496
đVitamin B12 āĻĻāĻŋāϝāĻŧā§ severe macrocytic (megaloblastic) anemia-āĻāϰ āĻāĻŋāĻāĻŋā§āϏāĻž āĻļā§āϰ⧠āĻāϰāĻžāϰ āĻĒāϰ Hypokalemia āĻšāϤ⧠āĻĒāĻžāϰā§āĨ¤
āĻāĻžāϰāĻŖ, Vitamin B12 āĻĻā§āĻāϝāĻŧāĻžāϰ āĻĢāϞ⧠bone marrow-āĻ āĻĻā§āϰā§āϤ red blood cell āĻā§āĻĒāĻžāĻĻāύ (erythropoiesis) āĻļā§āϰ⧠āĻšāϝāĻŧāĨ¤ āύāϤā§āύ āϤā§āϰāĻŋ āĻšāĻāϝāĻŧāĻž red blood cell-āĻā§āϞ⧠āϰāĻā§āϤ āĻĨā§āĻā§ āĻĒā§āϰāĻā§āϰ potassium āĻā§āϰāĻšāĻŖ āĻāϰā§, āϝāĻžāϰ āĻĢāϞ⧠āϏāĻžāĻŽāϝāĻŧāĻŋāĻāĻāĻžāĻŦā§ serum potassium-āĻāϰ āĻŽāĻžāϤā§āϰāĻž āĻāĻŽā§ āϝā§āϤ⧠āĻĒāĻžāϰā§āĨ¤ āϤāĻžāĻ severe anemia-āϤ⧠Vitamin B12 therapy āĻļā§āϰ⧠āĻāϰāĻžāϰ āĻĒāϰ āϰā§āĻā§āϰ serum potassium āύāĻŋāϝāĻŧāĻŽāĻŋāϤ monitor āĻāϰāĻž āĻāĻāĻŋāϤāĨ¤
6 496
25th edition davidson āĻ pneumothorax āĻāϰ āĻŽā§āϝāĻžāύā§āĻāĻŽā§āύā§āĻ āĻĒā§āϝāĻžāĻāĻžāϰā§āύ āĻā§āύā§āĻ āĻšā§ā§āĻā§:
đāĻĒā§āϰāĻĨāĻŽā§āĻ āĻĻā§āĻāϤ⧠āĻšāĻŦā§,āϰā§āĻā§ Symptomatic āύāĻžāĻāĻŋ Asymptomatic?
āĻāĻāĻžāĻ āĻĒā§āϰāĻĨāĻŽ āϏāĻŋāĻĻā§āϧāĻžāύā§āϤāĨ¤
āϝāĻĻāĻŋ Asymptomatic āĻšā§:
āϰā§āĻā§āϰ
* Chest pain āύā§āĻ āĻŦāĻž āĻā§āĻŦ āĻāĻŽ
* Dyspnea āύā§āĻ
* Stable
āϤāĻāύ....Size āϝāϤāĻ āĻšā§āĻ Conservative management āĻāϰāĻž āϝāĻžāϝāĻŧāĨ¤
Step 2: āϤāĻāύ āĻĻā§āĻāϤ⧠āĻšāĻŦā§:PSP āύāĻžāĻāĻŋ SSP?
Primary Spontaneous Pneumothorax (PSP)
Underlying lung disease āύā§āĻāĨ¤
âĄī¸ āϝāĻĻāĻŋ asymptomatic āĻšāϝāĻŧ
* āĻŦāĻžāĻĄāĻŧāĻŋ āĻĒāĻžāĻ āĻžāύ⧠āϝāĻžāĻŦā§
* Regular follow-up āĻ āϰāĻžāĻāϤ⧠āĻšāĻŦā§āĨ¤
đāĻāĻŋāύā§āϤā§,Secondary Spontaneous Pneumothorax (SSP)
Underlying lung disease āĻāĻā§
āϝā§āĻŽāύ
* COPD
* ILD
* Fibrosis
âĄī¸ Asymptomatic āĻšāϞā§āĻ
Hospital admission āĻāϰ⧠observation āĻāϰāϤ⧠āĻšāĻŦā§āĨ¤
đĨStep 3: āϝāĻĻāĻŋ Symptomatic āĻšāϝāĻŧ
āĻāĻāύ āĻĻā§āĻāϤ⧠āĻšāĻŦā§ High-risk features āĻāĻā§ āĻāĻŋāύāĻžāĨ¤
High-risk Features āĻā§ āĻā§ :
āϝā§āĻā§āύ⧠āĻāĻāĻāĻŋ āĻĨāĻžāĻāϞā§āĻ High-risk
â
Hemodynamic compromise
* Hypotension
* Shock
â
Hypoxia
â
Bilateral pneumothorax
â
Underlying lung disease
(COPD etc.)
â
Age >50 years + significant smoking history
â
Haemopneumothorax
āϝāĻĻāĻŋ High-risk āĻĨāĻžāĻā§
âĄī¸ Needle aspiration āύāϝāĻŧ
âĄī¸ āϏāϰāĻžāϏāϰāĻŋ Intercostal Chest Drain (ICD)
đāϝāĻĻāĻŋ High-risk āύāĻž āĻĨāĻžāĻā§
āϤāĻāύ āϤāĻŋāύāĻāĻŋ āĻ
āĻĒāĻļāύ
đOption 1
Percutaneous Needle Aspiration
â
Simple
â
Less invasive
â
ā§Ŧā§Ļâā§Žā§Ļ% āϰā§āĻā§āϰ Chest drain āϞāĻžāĻāĻŦā§ āύāĻžāĨ¤
đOption 2
Ambulatory device
One-way valve (Heimlich type)
â
āĻŦāĻžāĻĄāĻŧāĻŋāϤ⧠āĻĨā§āĻā§āĻ follow-up āĻāϰāĻž āϝāĻžāϝāĻŧāĨ¤
đOption 3
Conservative treatment
āϝāĻĻāĻŋ symptoms āĻāĻŽ āĻĨāĻžāĻā§ āĻāĻŦāĻ patient āϏāĻŽā§āĻŽāϤ āĻĨāĻžāĻā§āĨ¤
āĻāĻŋāύā§āϤā§,Needle aspiration āĻŦā§āϝāϰā§āĻĨ āĻšāϞā§?
āϝāĻĻāĻŋ
* āĻāĻāύāĻ dyspnea āĻĨāĻžāĻā§
* Pneumothorax persist āĻāϰā§
â
âĄī¸ Chest drain āϞāĻžāĻāĻŦā§āĨ¤
āϝāĻĻāĻŋ Tension Pneumothorax āĻšāϝāĻŧ:
āĻāĻāĻž EmergencyāĨ¤
immediate pressure release āĻāϰāϤ⧠neddle aspirantion āĻāϰāĻŦā§ āϤāĻžāϰāĻĒāϰ āĻĄā§āϰā§āĻāύ āĻāĻŋāĻāĻŦ āĻĻāĻŋāĻŦā§āĨ¤
đđđ Chest Drain āĻĻā§āĻāϝāĻŧāĻžāϰ āĻĒāϰā§
Drain connected āĻĨāĻžāĻāĻŦā§
â
Underwater seal āĻŦāĻž Heimlich valve-āĻāϰ āϏāĻžāĻĨā§āĨ¤
Drain āĻāĻāύ āĻā§āϞāĻŦā§?
āϝāĻāύ
âLung fully expanded
AND
âBubbling āĻŦāύā§āϧ
â
āĻĒāϰā§āϰ āĻĻāĻŋāύ Drain remove āĻāϰāĻž āϝāĻžāϝāĻŧāĨ¤
āϝāĻĻāĻŋ Bubbling āĻŦāύā§āϧ āύāĻž āĻšāϝāĻŧ?
āϝāĻĻāĻŋ
ā§Ēā§Ž āĻāĻŖā§āĻāĻžāϰ āĻŦā§āĻļāĻŋ bubbling āĻĨāĻžāĻā§
â
āĻŽāĻžāύā§
Persistent air leak
â
CT Chest āĻāϰāϤ⧠āĻšāĻŦā§
â
Thoracic surgeon-āĻā§ refer āĻāϰāϤ⧠āĻšāĻŦā§
â
āϏāĻŽā§āĻāĻŦāϤ Surgical repair āϞāĻžāĻāĻŦā§ (VATS/Bleb repair/Pleurodesis)āĨ¤
āϝāĻĻāĻŋ Bottle-āĻ bubbling āĻšāĻ āĻžā§ āĻŦāύā§āϧ āĻšāϝāĻŧā§ āϝāĻžāϝāĻŧ āĻāĻŋāύā§āϤ⧠Lung expand āĻšāϝāĻŧāύāĻŋ?
āĻāĻžāĻŦāϤ⧠āĻšāĻŦā§
* Tube blocked
* Tube kinked
* Tube displaced
āĻ
āϰā§āĻĨāĻžā§ Drain āĻ āĻŋāĻāĻŽāϤ⧠āĻāĻžāĻ āĻāϰāĻā§ āύāĻžāĨ¤
āĻāĻāĻŦāĻžāϰ āĻĒā§āϰ⧠āĻĢā§āϞā§āĻāĻžāϰā§āĻ āĻĻā§āĻā§āύ,āĻĒāĻžāύāĻŋāϰ āĻŽāϤ⧠āϏāĻšāĻ āĻšā§ā§ āϝāĻžāĻŦā§ āĻāύāĻļāĻžāĻāϞā§āϞāĻžāĻšāĨ¤
Dr Tamim
6 496
đĨanti tb āĻā§āϰāĻŋāĻāĻŽā§āύā§āĻ āĻļā§āϰ⧠āĻāϰāĻžāϰ ⧍ āϏāĻĒā§āϤāĻžāĻš āĻĒāϰ āĻĒā§āĻļā§āύā§āĻāĻā§
non infectious āϧāϰāĻž āϝā§āϤ⧠āĻĒāĻžāϰā§,āϝāĻĻāĻŋāύāĻž āĻā§āύ⧠āĻĄā§āϰāĻžāĻ resistance āĻĨāĻžāĻā§āĨ¤
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