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Welcome to My Telegram Channel. I'm sadhana singh. provide all basic Nursing information or pdf releted to disease or disorder conditions

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Lists of Phobia’s 1. Achluophobia – Fear of darkness 2. Acrophobia – Fear of heights 3. Aerophobia – Fear of flying 4. Algophobia – Fear of pain 5. Agoraphobia – Fear of open spaces or crowds 6. Aichimophobia – Fear of pointed objects 7. Amaxophobia – Fear of riding in a car 8. Androphobia – Fear of men 9. Anginophobia – Fear of angina or choking 10. Anthrophobia – Fear of flowers 11. Anthropophobia – Fear of people or society 12. Aphenphosmphobia – Fear of being touched. 13. Arithmophobia – Fear of numbers 14. Astraphobia – Fear of thunder and lightning 15. Ataxophobia – Fear of disorder or untidiness 16. Atelophobia – Fear of imperfection 17. Atychiphobia – Fear of failure 18. Autophobia – Fear of being alone 19. Bacteriophobia – Fear of bacteria 20. Barophobia – Fear of gravity21. Bathmophobia – Fear of stairs and steep slopes 22. Batrachophobia – Fear of amphibians 23. Belonephobia – Fear of pins and needles 24. Bibliophobia – Fear of books 25. Botanophobia – Fear of plants 26. Cacophobia – Fear of ugliness 27. Catagelophobia – Fear of being ridiculed 28. Catoptrophobia – Fear of mirrors 29. Chionophobia – Fear of snow 30. Chromophobia – Fear of colors 31. Chronomentrophobia – Fear of clocks 32. Claustrophobia – Fear of confined places 33. Coulrophobia – Fear of clowns 34. Cyberphobia – Fear of computers 35. Cynophobia – Fear of dogs 36. Dendrophobia – Fear of trees 37. Dentophobia – Fear of dentists 38. Domatophobia – Fear of houses 39. Dystychiphobia – Fear of accidents 40. Ecophobia – Fear of the home 41. Elurophobia – Fear of cats42. Entomphobia – Fear of insects 43. Ephebiphobia – Fear of teenagers 44. Equinophobia – Fear of horses 45. Gamophobia – Fear of marriage 46. Genuphobia – Fear of knees 47. Glossophobia – Fear of speaking in public 48. Gynophobia – Fear of women 49. Heliophobia – Fear of the sun 50. Hemophobia – Fear of blood 51. Herpetophobia – Fear of reptiles 52. Hydrophobia – Fear of water

IMPORTANT RULES in Medicine ✨ Rule of HALVES – Hypertension & Diabetes • Only 50% of individuals with hypertension or diabetes are diagnosed. • Of those diagnosed, only 50% receive treatment. • Of those treated, only 50% achieve control of their condition. • This rule highlights the gaps in diagnosis, treatment, and management. āø» ✨ Rule of 5 – Pregnancy • This rule is related to the size of the uterus during pregnancy: • 5 weeks – Uterus is just palpable. • 10 weeks – Uterus reaches the pubic symphysis. • 15 weeks – Uterus is midway between the pubic symphysis and umbilicus. • 20 weeks – Uterus reaches the umbilicus. • 25 weeks and beyond – Uterus continues to grow progressively. āø» ✨ Rule of 9 – Burns • Used to estimate Total Body Surface Area (TBSA) affected by burns: • Head & Neck – 9% • Each Arm – 9% (4.5% front, 4.5% back) • Each Leg – 18% (9% front, 9% back) • Front of Torso – 18% • Back of Torso – 18% • Perineum – 1% • Helps guide fluid resuscitation and management. āø» ✨ Rule of 10 – Cleft Lip & Palate • Used to determine optimal timing for cleft lip repair (Rule of 10s): • 10 weeks old • 10 pounds (4.5 kg) weight • Hemoglobin of 10 g/dL • Ensures the baby is healthy enough for surgery. āø» ✨ Rule of 12 – Rigor Mortis • Refers to the onset and resolution of rigor mortis in a 12-hour cycle: • First 12 hours – Rigor mortis develops. • Next 12 hours – Rigor mortis is fully established. • Final 12 hours – Rigor mortis disappears. • Used in forensic medicine to estimate time of death. āø» ✨ Rule of 15 – Hypoglycemia • Used to manage hypoglycemia (low blood sugar levels): • If blood glucose is ≤70 mg/dL, give 15g of fast-acting carbohydrate (e.g., glucose tablets, juice). • Wait 15 minutes and recheck blood glucose. • If still low, repeat 15g of glucose and recheck after another 15 minutes. • Once stable, eat a small meal or snack to prevent recurrence.

PORTAL HYPERTENSION Normal portal vein pressure is 5-10 mm of Hg. Poratl hypertension is defined as an elevation of portalvenous pressure> 10-12 mm of Hg It is most common cause of Gastroi.

# Infection control Central line infections can be prevented by following proper hygiene practices and using antiseptics. Patients and their families can also ask their healthcare provider about their options and how to care for the line. Patient practices :- * Ask your healthcare provider to clean their hands and wear gloves before touching the catheter * Avoid touching the tubing or where it exits the skin * Ask your healthcare provider if you still need the catheter * Remove the catheter as soon as it's no longer needed Healthcare provider practices :- * Use chlorhexidine or povidone iodine to clean the catheter opening * Use sterile devices to access catheters * Change dressings regularly, especially if they're wet or soiled * Use aseptic technique when changing dressings * Use the minimum number of ports or lumens needed Other practices :- * Wash hands before entering and leaving a patient's room * Follow recommended infection control guidelines * Use full-barrier precautions when inserting the catheter * Avoid using the femoral vein for adult patients

ANATOMY LENGTHS 4cm long structures in the body šŸ‘‰Inguinal canal šŸ‘‰Anal canal šŸ‘‰Female urethra šŸ‘‰Auditory tube šŸ‘‰Cystic duct šŸ‘‰Optic nerve šŸ‘‰Prostatic urethra šŸ‘‰Left principle bronchus 5cm long structures in the body šŸ‘‰Male larynx šŸ‘‰Left primary bronchus šŸ‘‰Parotid duct šŸ‘‰Submandibular duct šŸ‘‰Lateral lobe of thyroid gland šŸ‘‰Testis šŸ‘‰First part of duodenum šŸ‘‰Neck of femur šŸ‘‰Lateral wall of bony orbit šŸ‘‰Medial wall of bony orbit 7.5cm long structures in the body šŸ‘‰Anterior wall of vagina šŸ‘‰Virgin uterus šŸ‘‰Second part of duodenum šŸ‘‰Bile duct 9cm long structures in the body šŸ‘‰Appendix šŸ‘‰Multiparous uterus šŸ‘‰Posterior wall of vagina 11cm long structures in the body šŸ‘‰Trachea šŸ‘‰Kidney šŸ‘‰Uterine tube šŸ‘‰Third part of duodenum 12cm long structures in the body šŸ‘‰Rectum šŸ‘‰Spleen šŸ‘‰Pharynx 15cm long structures in the body šŸ‘‰Adductor canal šŸ‘‰Ascending colon šŸ‘‰Pancreas šŸ‘‰Root of mesentery 25cm long structures in the body šŸ‘‰Oesophagus šŸ‘‰Descending colon šŸ‘‰Ureter šŸ‘‰Duodenum 45cm long structures in the body šŸ‘‰Spinal cord šŸ‘‰Thoracic duct šŸ‘‰Transverse colon šŸ‘‰Femur šŸ‘‰Sartorius šŸ‘‰Vas deferens

ā˜…Father of Biology: *Aristotle* ā˜…Father of Physics: *Albert Einstein* ā˜…Father of Chemistry: *Jabir Bin Hayan* ā˜…Father of Statistics: *Ronald Fisher* ā˜…Father of Zoology: *Aristotle* ā˜…Father of History: *Herodotus* ā˜…Father of Microbiology: *Louis Pasteur* ā˜…Father of Botany: *Theophrastus* ā˜…Father of Algebra: *Muhammad ibn Musa al-Khwarizmi* ā˜…Father of Blood groups: *Landsteiner* ā˜…Father of Electricity: *Benjamin Franklin* ā˜…Father of Trigonometry: *Hipparchus* ā˜…Father of Geometry: *Euclid* ā˜…Father of Modern Chemistry: *Antoine Lavoisier* ā˜…Father of Robotics: *Engelberger* ā˜…Father of Electronics: *Michael Faraday* ā˜…Father of Internet: *Vinton Cerf* ā˜…Father of Economics: *Adam Smith* ā˜…Father of Video game: *Ralph Baer* ā˜…Father of Architecture: *Louis Henry Sullivan* ā˜…Father of Genetics: *Gregor Johann Mendel* ā˜…Father of Nanotechnology: *Heinrich Rohrer* ā˜…Father of C language: *Dennis Ritchie* ā˜…Father of World Wide Web: *Tim Berners-Lee* ā˜…Father of Search engine: *Alan Emtage* ā˜…Father of Periodic table: *Dmitri Mendeleev* ā˜…Father of Taxonomy: *Carolus Linnaeus* ā˜…Father of Surgery (early): *Sushruta* ā˜…Father of Mathematics: *Archimedes* ā˜…Father of Medicine: *Hippocrates* ā˜…Father of Homeopathy: *Samuel Hahnemann*...!

*CVP Line* • Preferred site of CVP insertion: *Right internal jugular vein* • M/C site of insertion for TPN administration: *Subclavian vein* • M/C site with least risk of infection: *Subclavian vein* • M/C site with high risk of infection and DVT: *Femoral vein* • M/C site in emergency: *Femoral Vein* • High risk of pneumothorax: *Subclavian* • Patient position while inserting the CVP line through jugular or subclavian vein: *Trendelenburg* • M/C site of insertion of PICC line: *Basilic vein* • Technique used to insert the CVAC: *Seldinger technique* • Solution for cleaning insertion site: *2% CHG*

Nutritional programme āœļøMid-Day Meal Programme -1961 āœļøSchool Health Programme - 1962 āœļøNational Goiter control programme -1962 āœļøApplied Nutrition Programme -1963 āœļøVit-A Prophylaxis programme - 1970 āœļøSpecial Nutrition Programme -1970 āœļøBalwadi Nutrition Programme -1970 āœļøMid day meal Scheme -15 Aug 1995

*-Disposable Of PPE* 1. *Head Cover:* Yellow 🟔 2. *Goggle:* Red šŸ”“ 3. *Face Shield:* Red šŸ”“ 4. *Surgical Mask:* Yellow 🟔 5. *N-95 Mask:* Yellow 🟔 6. *Surgical Gown:* Yellow 🟔 7. *Isolation Grown:* Yellow 🟔 8. *Coverall Suits:* Yellow 🟔 9. *Gloves:* Red šŸ”“ 10. *Shoes:* Yellow 🟔

_šŸ”¹TUBERCULOSIS CASE MANAGEMENT_ _šŸ”¹DIAGNOSIS OF TB_ ā–ŖļøThe presence of pulmonary tuberculosis should be suspected in individuals presenting with one or more of the following complaints: ā–ŖļøCough for 1 week or longer, ā–ŖļøNight sweats ā–ŖļøFever ā–ŖļøLoss of weight, ā–ŖļøA body mass index(BMI) <17kg/m2 or failure to thrive among children. _šŸ”¹LABORATORY INVESTIGATIONS_ ā–ŖļøEvery effort should be made to bacteriologically confirm the diagnosis of TB,with rapid molecular test such as the Xpert MTB/Rif test being the preferred diagnostic test for patients being evaluated for tuberculosis. ā–Ŗļø A specimen should be submitted for all cases being investigated for pulmonary TB as per TB diagnostic algorithm, in order to confirm the diagnosis of Tuberculosis. _šŸ”¹Chest X-RAYS_ ā–ŖļøChest X-ray is recommended as a priority SCREENING test for persons classified as being at high risk for tuberculosis. ā–ŖļøThe following persons are classified as being at high risk for tuberculosis: ā–«ļøChildren under the age of 5 years. ā–«ļøPeople living with HIV. ā–«ļøThose who are malnourished. ā–«ļøPeople above 60 years of age. ā–«ļøPeople with diabetes mellitus. ā–«ļøPeople who drink alcohol excessively ā–«ļøMiners and ex-miners ā–«ļøPeople in congregate settings. ā–«ļøInmates and correctional facility communities. ā–«ļøHealth care workers ā–ŖļøNote: In the presence of clinical improvement, it is not necessary to monitor the response of pulmonary TB to treatment by chest x-rays. _TUBERCULIN TESTING_ ā–Ŗļø0.1 mls intradermal ā–ŖļøExamine induration at 48-72 hours. ā–ŖļøA positive tuberculin skin test (person with normal immunity: induration > 10 mm, person with defective immunity: induration > 5 mm) may indicate active infection (especially if strongly positive) , previous infection or previous BCG. ā–ŖļøAbsence of a response does not exclude TB because individuals with HIV may not have sufficient immunity for a positive skin test despite active TB. ā–ŖļøIf a child under 3 years of age has not had BCG,the Mantoux test may be useful. _šŸ”¹TREATMENT REGIMEN FOR TB_ ā–ŖļøOne treatment regimen is now used in Zimbabwe for the treatment of drug sensitive TB,regardless of the treatment history. ā–ŖļøThe regimen consists of a combination of the four first line medicines HRZE: H= isoniazid R= rifampicin Z= pyrazinamide E= ethambutol ā–ŖļøThese medicines are available as Fixed Dose Combination (FDC). ā–ŖļøThe intention of these combination tablets is to improve compliance by reducing the number of tablets a patient takes, and to reduce the possibility of medicine resistance developing. ā–ŖļøThe number of FDC tablets is determined by a weight range of each patient at the start of treatment ā–ŖļøTreatment is the same for HIV infected people as for non-HIV _šŸ”¹TREATMENT OF DRUG-SENSITIVE TB_ ā–ŖļøAll drug sensitive cases of TB regardless of site, severity or previous treatment history are treated using the regimens summarised below.

_šŸ”¹BONE HEALING_ ā–ŖļøBone healing, also known as bone fracture repair,is a complex and dynamic process by which the body restores damaged or fractured bone tissue to its original strength and function. ā–ŖļøThe process of bone healing involves several stages: _1.FRACTURE HEMATOMA_ ā–ŖļøWhen a fracture occurs,bleeding creates a hematoma,which surrounds the ends of the fragments. ā–ŖļøThe hematoma is an extravasated blood that changes from a liquid to a semisolid clot. ā–ŖļøThis occurs in the initial 72 hours after injury. _2.GRANULATION TISSUE_ ā–ŖļøDuring this stage,active phagocytosis absorbs the products of local necrosis. ā–ŖļøThe hematoma converts to granulation tissue. ā–ŖļøGrannulation tissue(consisting of new blood vessels,fibroblasts and osteoblasts) produces the basis for new bone substance called osteoid during 3 days to 14 days post injury. _3.CALLUS FORMATION_ ā–ŖļøAs minerals (calcium,phosphorus,and magnesium) and new bone matrix are deposited in the osteoid,an organized network of bone is formed that is woven about the fracture parts. ā–ŖļøCallus is primarily composed of cartilage,osteoblasts,calcium and phosphorus. ā–ŖļøIt usually appears by the end of the second week after injury. ā–ŖļøEvidence of callus formation can be verified by x-ray _4.OSSIFICATION_ ā–ŖļøOccurs from 3 weeks to 6 months after the fracture and continues until the fracture has healed. ā–ŖļøCallus ossification is sufficient to prevent movement at the fracture site when the bones are gently stressed. ā–ŖļøHowever,the fracture is still evident on x-ray. ā–ŖļøDuring this stage of clinical union,the patient may be allowed limited mobility or the cast may be removed. _5.CONSOLIDATION_ ā–ŖļøAs callus continues to develop,the distance between bone fragments diminishes and eventually closes. ā–ŖļøDuring this stage ossification continues. ā–ŖļøIt can be equated with radiological union,which occurs when there is x-ray evidence of complete bone union. ā–ŖļøThis phase occur up to 1 year after injury _6.REMODELING_ ā–ŖļøExcess bone tissue is resorbed in the final stage of bone healing, ane union is complete. ā–ŖļøGradual return of injured bone to its preinjury structural strength and shape occurs. ā–ŖļøBone remodels in response to physical loading stress or wolf's law.Initially stress is provided through exercise.

*Amlodipine* *GROUP:* Antihypertensive *CLASS:* Calcium Channel Blockers(CCBs) _šŸ”¹MODE OF ACTION_ ā–ŖļøIt inhibits the influx(entry) of calcium ions into vascular smooth muscle cells, leading to relaxation and vasodilation. ā–ŖļøThis vasodilation decreases peripheral resistance, resulting in a decrease in blood pressure. _šŸ”¹ROLE OF CALCIUM IONS IN HUMAN BODY_ ā–ŖļøCalcium is necessary for muscle contractions to occur throughout the body. ā–Ŗļø This mineral enters muscle cells through various ion channels (L,N,T,P and R),which are tiny pores on the surface of the cell. ā–ŖļøThis process is essential for normal body functioning. ā–ŖļøCCB's reduce the amount of calcium that can enter muscle cells in the heart and blood vessel walls via L-type calcium-channels. ā–ŖļøThis lowers the pressure in the blood vessels and on the heart. _šŸ”¹PRESENTATION_ ā–ŖļøTablets;2,5mg,5mg,10mg ā–ŖļøCapsules;5mg,10mg _šŸ”¹INDICATIONS_ ā–ŖļøHypertension ā–ŖļøAngina pectoris _šŸ”¹CONTRAINDICATIONS & CAUTIONS_ ā–ŖļøContraindicated in patients hypersensitive to drug. ā–ŖļøUse cautiously in patients receiving other peripheral vasodilators, especially those with severe aortic stenosis, and in those with heart failure. ā–ŖļøBecause drug is metabolized by the liver,use cautiously and in reduced dosage in patients with severe hepatic disease. _šŸ”¹DOSE,ROUTE,FREQUENCY_ ā–ŖļøAdults:5-10 mg per oral once daily as long term. ā–ŖļøPaeds:2.5mg - 5mg per oral once daily _šŸ”¹HALF-LIFE_ ā–Ŗļø30 to 50 hours. _šŸ”¹ADVERSE REACTIONS_ _šŸ”¹CENTRAL NERVOUS SYSTEM_ ā–Ŗļø Headache. ā–ŖļøSomnolence. ā–ŖļøDizziness. ā–ŖļøLight-headedness. ā–ŖļøAsthenia. _šŸ”¹CARDIOVASCULAR SYSTEM_ ā–ŖļøOedema. ā–ŖļøFlushing. ā–ŖļøPalpitations. _šŸ”¹GASTROINTESTINAL_ ā–ŖļøDyspepsia. ā–Ŗļø Nausea. ā–ŖļøAbdominal pain. _šŸ”¹GENITOURINARY_ ā–ŖļøSexual difficulties. _šŸ”¹MUSCULOSKELETAL_ ā–ŖļøMuscle cramps. _šŸ”¹RESPIRATORY_ ā–ŖļøDyspnea. _šŸ”¹SKIN_ ā–ŖļøRash. ā–ŖļøPruritus. _šŸ”¹INTERACTIONS_ ā–ŖļøNone reported. _šŸ”¹EFFECTS ON LAB TEST RESULTS_ ā–ŖļøNone reported. _šŸ”¹OVERDOSE S&S:_ ā–ŖļøHypotension. _šŸ”¹NURSING CONSIDERATIONS_ ā–ŖļøMonitor patient carefully.Some patients,especially those with severe obstructive coronary artery disease,have developed increased frequency,duration,or severity of angina or acute Myocardial infarction after initiation of calcium channel blocker therapy or at time of dosage increase. ā–ŖļøMonitor blood pressure frequently during initiation of therapy because drug-induced vasodilation has a gradual onset,acute hypotension is rare. ā–ŖļøAbrupt withdrawal of drug may increase frequency and duration of chest pain.Taper dose gradually under medical supervision. ā–ŖļøLook alike–sound alike: Don’t confuse amlodipine with amiloride. _šŸ”¹PATIENT TEACHING_ ā–ŖļøCaution patient to continue taking drug,even when he feels better. ā–ŖļøTell patient that nitroglycerin may be taken as needed when angina symptoms are acute.If patient continues nitrate therapy during adjustment of amlodipine dosage,urge continued compliance.

*_Native valve endocarditis_* ā–ŖļøBenzylpenicilin 5MU 6 hourly 2- 6 weeks. ā–ŖļøPaeds dose:0.05-0.1MU/kg ā–Ŗļø Gentamycin 80-120mg IV 12 hourly for 2 weeks. ā–ŖļøPaeds dose: 1-1.5 mg/kg. ā–ŖļøCeftriaxone 1g IV 12hrly for 2-6 weeks. ā–ŖļøPaeds dose:50mg/kg *_Prosthetic valve endocarditis_* ā–ŖļøCloxacillin 2g 6hrly for 4-6 weeks. ā–ŖļøPaeds dose:15mg/kg ā–ŖļøGentamycin 80-120mg 12hrly for 4 weeks. ā–ŖļøPaeds dose:1-1.5mg/kg *_Treatment of culture positive endocarditis_* ā–ŖļøBenzylpenicilin 5MU IV 6hrly for 4-6 weeks. ā–ŖļøGentamycin 80-120mg 12hrly for 4 weeks. _šŸ”¹COMPLICATIONS_ ā–ŖļøValvular heart disease. ā–Ŗļø Heart failure. ā–ŖļøMyocarditis.

_šŸ”¹ENDOCARDITIS_ ā–ŖļøEndocarditis is an inflammation of the inner layer of the heart. ā–ŖļøIt is usually caused by bacteria, fungi, or viruses entering the bloodstream through invasive procedures like dental work or surgery. ā–ŖļøThe microbes accumulate on the heart valves, forming vegetations that can damage the valves over time. _šŸ”¹CAUSES_ *_(a)Bacteria._* ā–ŖļøStreptococcus viridan. ā–ŖļøStaphylococcus Aureus. _*(b)Fungi*_ ā–ŖļøAspagellus ā–ŖļøCandida *_(c).Acute rheumatic fever which causes enlarged and tender lymph nodes,damages the valves._* _*(d).Congenital heart disease.*_ _šŸ”¹RISK FACTORS_ ā–ŖļøPrevious heart damage. ā–ŖļøHeart surgeries. ā–ŖļøDental surgeries can introduce bacterias. ā–ŖļøLong term haemodialysis. ā–ŖļøDiabetes Mellitus. *_šŸ”¹TYPES_* _*(I)ACUTE ENDOCARDITIS*_ ā–ŖļøInfections caused by staphylococcus Aureus occurs rapidly on normal valves. _*(II)SUBACUTE ENDOCARDITIS*_ ā–ŖļøThis is infection caused by streptococcus viridans and usually develops gradual on previously damaged heart valves e.g. rheumatic heart disease. *_(III)POST OPERATIVE ENDOCARDITIS_* ā–Ŗļø Infection following cardiac surgery in which prosthetic heart valves are used. ā–Ŗļø It is caused by staphylococcus. *_šŸ”¹PATHOPHYSIOLOGY_* ā–ŖļøA deformity or injury of the endocardium leads to accumulation fibrin and platelets (clot formation). ā–ŖļøThe infection most frequently results in platelets,fibrin,blood cells,and microorganisms that clusteras vegetations on the endocardium. ā–ŖļøThe vegetations may embolize to other tissues throughout the body. ā–ŖļøAs the clot on the endocardium continues to expand,the infecting organism is covered by the new clot and concealed from the body's normal defenses. ā–ŖļøThe infection may erode through the endocardium into the underlying structures(eg,valve leaflets),causing tears or other deformities of valve leaflets,dehiscence of prosthetic valves, deformity of the chordae tendineae,or mural abscesses. ā–ŖļøUsually the onset of infective endocarditis is insidious. ā–ŖļøThe signs and symptoms develop from the toxic effect of the infection,from destruction of the heart valves,and from embolization of fragments of vegetative growths on the heart. ā–ŖļøSystemic emboli occur with lef-sided heart infective endocardis;pulmonary emboli occur with right-sided heart infective endocarditis. _OR_ ā–ŖļøThe endocardium is invaded by microbes directly. ā–ŖļøThe microbes cause deformity, thickening, stiffening and scaring of the valve leaflets and may travel to other organs and obstruct blood flow. ā–Ŗļø This results in valvular incompetence and invasion of the myocardium by infection resulting myocardial dysfunction, sepsis and congestive cardiac failure. _šŸ”¹CLINICAL FEATURES_ ā–ŖļøFever. ā–ŖļøTachycardia ā–ŖļøPetechiae conjunctiva. ā–ŖļøSplinter hemorrhages in nail beds. ā–ŖļøOsler's nodes -painful red nodes on pads of fingers and toes; usually late sign of infection and found with a sub acute infection. ā–ŖļøMurmurs with other signs and symptoms may indicate right side heart infection. ā–ŖļøTachycardia related to decreased cardiac output. ā–ŖļøLocalized headaches. ā–ŖļøTransient cerebral ischemia. ā–Ŗļø Altered mental status, aphasia. ā–ŖļøHemiplegia. ā–Ŗļø Cortical sensory loss ā–ŖļøHaemoptysis. ā–ŖļøChest pain, shortness of breath. ā–ŖļøKidney --haematuria. ā–ŖļøSpleen- pain in left upper quadrant of abdomen radiating to left shoulder. _šŸ”¹DIAGNOSTIC EVALUATIONS_ ā–ŖļøBlood cultures for isolation of the causative organism. ā–ŖļøEchocardiogram for identification of infected area. ā–Ŗļø Blood for white cell count and erythrocyte sedimentation rate which show elevated levels. ā–ŖļøCXR to detect cardiomegaly. ā–ŖļøUrinalysis to see microscopic hematuria. ā–ŖļøComputerized tomography rule out heart damage ā–ŖļøReview bun and creatinine levels to evaluate renal function. ā–ŖļøReview endocardiography finding if available for valvuler and ventricular function and presence of vegetation. _šŸ”¹MANAGEMENT_

Here are some common diseases related to the digestive system: 1. Gastroesophageal Reflux Disease (GERD) 2. Inflammatory Bowel Disease (IBD) - Crohn's disease - Ulcerative colitis 3. Irritable Bowel Syndrome (IBS) 4. Peptic Ulcer Disease 5. Diverticulitis 6. Gastrointestinal Bleeding 7. Celiac Disease 8. Chronic Pancreatitis 9. Gallstones 10. Cholecystitis (inflammation of the gallbladder) 11. Hepatitis (liver inflammation) 12. Cirrhosis (liver scarring) 13. Liver Cancer 14. Gastrointestinal Stromal Tumors (GISTs) 15. Colon Cancer 16. Rectal Cancer 17. Anal Cancer 18. Dysphagia (swallowing difficulty) 19. Achalasia (esophageal motility disorder) 20. Scleroderma (systemic sclerosis) These diseases can be categorized into various groups, such as: - Inflammatory conditions (e.g., IBD, celiac disease) - Functional disorders (e.g., IBS, dyspepsia) - Structural disorders (e.g., peptic ulcer disease, diverticulitis) - Neoplastic disorders (e.g., colon cancer, GISTs) - Infections (e.g., hepatitis, gastroenteritis) - Motility disorders (e.g., achalasia, scleroderma) It's important to note that this is not an exhaustive list, and there are many other diseases and conditions that can affect the digestive system.

*Actinic Keratosis (AK)* - A precancerous skin condition caused by prolonged exposure to UV radiation from the sun or tanning beds - Characterized by rough, scaly, or crusty patches on sun-exposed areas (face, ears, hands, arms, and legs) - Can progress to squamous cell carcinoma (SCC) if left untreated - Typically affects fair-skinned individuals over 40 years old - Treatment options include: - Topical creams or gels (e.g., fluorouracil, imiquimod) - Cryotherapy (freezing) - Photodynamic therapy (PDT) - Surgical excision - Prevention: sun protection (sunscreen, clothing, hats), avoid tanning beds, and regular skin checks Early detection and treatment can prevent progression to SCC and reduce the risk of complications.

⚔ Mirena is a hormonal intrauterine device (IUD) that provides long-term contraception and is approved for the treatment of heavy menstrual bleeding (menorrhagia). It releases a small amount of the hormone levonorgestrel, which helps reduce menstrual flow and prevent pregnancy for up to 5-8 years. ⚔Minipill : A progestin-only oral contraceptive, not an IUD. ⚔ Copper T : A non-hormonal IUD that provides long-term contraception but does not treat menorrhagia and may worsen bleeding. ⚔ Norplant (Subdermal) : A set of hormone-releasing implants placed under the skin for contraception but not specifically used to treat menorrhagia.

Hyperphosphatemia (elevated levels of phosphate in the blood) significantly affects the heart, especially in individuals with chronic kidney disease (CKD) or other metabolic disorders. Here’s the pathophysiology: 1. Vascular Calcification • High phosphate levels lead to vascular smooth muscle cell (VSMC) transformation into osteoblast-like cells. • This triggers deposition of calcium-phosphate in the arterial walls, resulting in vascular calcification. • Calcification reduces the flexibility of blood vessels, increasing vascular stiffness and contributing to hypertension and increased afterload on the heart. 2. Cardiac Calcification • Hyperphosphatemia can also lead to calcification of the heart valves (e.g., aortic and mitral valves) and myocardium. • Valve calcification can impair their function, causing stenosis or regurgitation, which increases the workload of the heart and predisposes to heart failure. 3. Endothelial Dysfunction • Elevated phosphate disrupts endothelial cell function, reducing nitric oxide (NO) production and promoting oxidative stress. • This contributes to arterial stiffness, increased vascular resistance, and heightened risk of atherosclerosis. 4. Left Ventricular Hypertrophy (LVH) • Increased vascular resistance and calcification elevate cardiac workload, causing left ventricular hypertrophy. • LVH is associated with poor cardiac output and increased risk of arrhythmias, heart failure, and sudden cardiac death. 5. Secondary Hyperparathyroidism • In CKD, hyperphosphatemia often coexists with secondary hyperparathyroidism due to disrupted calcium-phosphate balance. • Elevated parathyroid hormone (PTH) levels further promote bone resorption and calcium-phosphate deposition in tissues, worsening vascular calcification and cardiac dysfunction. 6. Pro-Inflammatory State • Hyperphosphatemia induces systemic inflammation, which exacerbates atherosclerosis and impairs cardiac function. Clinical Implications Hyperphosphatemia is a major risk factor for cardiovascular morbidity and mortality, especially in CKD patients. Managing phosphate levels through dietary restrictions, phosphate binders, and maintaining calcium-phosphate balance is crucial to prevent or mitigate these cardiac complications.

MEDICAL TERMINOLOGIES 1. Spinal fusion - Surgical joining of vertebrae 2. Splenomegaly - Enlarged spleen 3. Spondylitis - Inflammation of spine 4. Spondylosis - Degeneration of spine 5. Squamous cell carcinoma - Skin cancer 6. Stenosis - Narrowing of blood vessel 7. Sterilization - Germ killing 8. Stethoscope - Heart and lung examination device 9. Stomatitis - Mouth inflammation 10. Stroke - Brain attack 11. Subarachnoid hemorrhage - Bleeding in brain 12. Subcutaneous - Under the skin 13. Sudden infant death syndrome (SIDS) - Unexplained infant death 14. Suicide - Intentional self-harm 15. Sulfonamide - Antibacterial medication 16. Superficial - Near the surface 17. Supination - Rotating upward 18.Supraventricular tachycardia - Fast heartbeat 19. Surgery - Operation 20. Suture - Stitch 21. Sympathectomy - Nerve removal 22. Symptom - Disease sign 23. Syncope - Fainting 24. Syndactyly - Fused fingers or toes 25. Syndrome - Group of symptoms 26. Synovitis - Joint inflammation 27. Systemic lupus erythematosus (SLE) - Autoimmune disorder 28. Tachycardia - Fast heartbeat 29. Tachypnea - Rapid breathing 30. Talipes equinovarus (TEV) - Clubfoot 31. Tamoxifen - Breast cancer medication 32. Tardive dyskinesia - Movement disorder 33. Tarsal tunnel syndrome - Foot nerve compression 34. Tendonitis - Tendon inflammation 35. Tenosynovitis - Tendon sheath inflammation 36. Testicular torsion - Twisted testicle 37. Thalassemia - Blood disorder 38. Thallium - Heavy metal poisoning 39. Thoracentesis - Chest fluid removal 40. Thrombocytopenia - Low platelet count 41. Thrombolysis - Blood clot dissolution 42. Thrombophlebitis - Vein inflammation 43. Thrombosis - Blood clot 44. Thyroid crisis - Thyroid storm 45. Thyroiditis - Thyroid inflammation 46. Tinnitus - Ringing in ears 47. Tonsillitis - Tonsil inflammation 48. Topical - Applied to skin 49. Torsion - Twisting 50. Torticollis - Twisted neck 51. Total parenteral nutrition (TPN) - Intravenous nutrition 52. Toxemia - Poisoning 53. Toxoplasmosis - Parasitic infection 54. Tracheitis - Windpipe inflammation 55. Tracheostomy - Breathing tube 56. Trachoma - Eye infection 57. Transfusion - Blood transfer 58. Transient ischemic attack (TIA) - Mini-stroke 59. Transplant - Organ replacement 60. Trauma - Injury 61. Tremor - Shaking 62. Trichomoniasis - Vaginal infection 63. Triglyceride - Blood fat 64. Triiodothyronine (T3) - Thyroid hormone 65. Troponin - Heart attack marker 66. Tuberculosis (TB) - Infection 67. Tumor - Abnormal growth 68. Tympanic membrane - Eardrum 69. Tympanitis - Middle ear inflammation 70. Tyrosinemia - Genetic disorder 71. Ulcer - Wound 72. Ultrasound - Imaging test 73. Unconscious - Comatose 74. Ureters - Kidney tubes 75. Urethra - Urinary tube 76. Uric acid - Waste product 77. Urinalysis - Urine examination 78. Urology - Urinary specialty 79. Uterine fibroids - Tumors in uterus 80. Uveitis - Eye inflammation 81. Vagina - Female genital canal 82. Vaginitis - Vaginal inflammation 83. Vagotomy - Vagus nerve cutting 84. Valvuloplasty - Heart valve repair 85. Varicella - Chickenpox 86. Varicocele - Enlarged veins in scrotum 87. Vasculitis - Blood vessel inflammation 88. Vasodilation - Blood vessel widening 89. Vasopressor - Blood pressure medication 90. Venereal disease - Sexually transmitted disease

Lists of Phobia’s 1. Achluophobia – Fear of darkness 2. Acrophobia – Fear of heights 3. Aerophobia – Fear of flying 4. Algophobia – Fear of pain 5. Agoraphobia – Fear of open spaces or crowds 6. Aichimophobia – Fear of pointed objects 7. Amaxophobia – Fear of riding in a car 8. Androphobia – Fear of men 9. Anginophobia – Fear of angina or choking 10. Anthrophobia – Fear of flowers 11. Anthropophobia – Fear of people or society 12. Aphenphosmphobia – Fear of being touched. 13. Arithmophobia – Fear of numbers 14. Astraphobia – Fear of thunder and lightning 15. Ataxophobia – Fear of disorder or untidiness 16. Atelophobia – Fear of imperfection 17. Atychiphobia – Fear of failure 18. Autophobia – Fear of being alone 19. Bacteriophobia – Fear of bacteria 20. Barophobia – Fear of gravity21. Bathmophobia – Fear of stairs and steep slopes 22. Batrachophobia – Fear of amphibians 23. Belonephobia – Fear of pins and needles 24. Bibliophobia – Fear of books 25. Botanophobia – Fear of plants 26. Cacophobia – Fear of ugliness 27. Catagelophobia – Fear of being ridiculed 28. Catoptrophobia – Fear of mirrors 29. Chionophobia – Fear of snow 30. Chromophobia – Fear of colors 31. Chronomentrophobia – Fear of clocks 32. Claustrophobia – Fear of confined places 33. Coulrophobia – Fear of clowns 34. Cyberphobia – Fear of computers 35. Cynophobia – Fear of dogs 36. Dendrophobia – Fear of trees 37. Dentophobia – Fear of dentists 38. Domatophobia – Fear of houses 39. Dystychiphobia – Fear of accidents 40. Ecophobia – Fear of the home 41. Elurophobia – Fear of cats42. Entomphobia – Fear of insects 43. Ephebiphobia – Fear of teenagers 44. Equinophobia – Fear of horses 45. Gamophobia – Fear of marriage 46. Genuphobia – Fear of knees 47. Glossophobia – Fear of speaking in public 48. Gynophobia – Fear of women 49. Heliophobia – Fear of the sun 50. Hemophobia – Fear of blood 51. Herpetophobia – Fear of reptiles 52. Hydrophobia – Fear of water