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Lists of Phobiaās
1. Achluophobia ā Fear of darkness
2. Acrophobia ā Fear of heights
3. Aerophobia ā Fear of flying
4. Algophobia ā Fear of pain
5. Agoraphobia ā Fear of open spaces or crowds
6. Aichimophobia ā Fear of pointed objects
7. Amaxophobia ā Fear of riding in a car
8. Androphobia ā Fear of men
9. Anginophobia ā Fear of angina or choking
10. Anthrophobia ā Fear of flowers
11. Anthropophobia ā Fear of people or society
12. Aphenphosmphobia ā Fear of being touched.
13. Arithmophobia ā Fear of numbers
14. Astraphobia ā Fear of thunder and lightning
15. Ataxophobia ā Fear of disorder or untidiness
16. Atelophobia ā Fear of imperfection
17. Atychiphobia ā Fear of failure
18. Autophobia ā Fear of being alone
19. Bacteriophobia ā Fear of bacteria
20. Barophobia ā Fear of gravity21. Bathmophobia ā Fear of stairs and steep slopes
22. Batrachophobia ā Fear of amphibians
23. Belonephobia ā Fear of pins and needles
24. Bibliophobia ā Fear of books
25. Botanophobia ā Fear of plants
26. Cacophobia ā Fear of ugliness
27. Catagelophobia ā Fear of being ridiculed
28. Catoptrophobia ā Fear of mirrors
29. Chionophobia ā Fear of snow
30. Chromophobia ā Fear of colors
31. Chronomentrophobia ā Fear of clocks
32. Claustrophobia ā Fear of confined places
33. Coulrophobia ā Fear of clowns
34. Cyberphobia ā Fear of computers
35. Cynophobia ā Fear of dogs
36. Dendrophobia ā Fear of trees
37. Dentophobia ā Fear of dentists
38. Domatophobia ā Fear of houses
39. Dystychiphobia ā Fear of accidents
40. Ecophobia ā Fear of the home
41. Elurophobia ā Fear of cats42. Entomphobia ā Fear of insects
43. Ephebiphobia ā Fear of teenagers
44. Equinophobia ā Fear of horses
45. Gamophobia ā Fear of marriage
46. Genuphobia ā Fear of knees
47. Glossophobia ā Fear of speaking in public
48. Gynophobia ā Fear of women
49. Heliophobia ā Fear of the sun
50. Hemophobia ā Fear of blood
51. Herpetophobia ā Fear of reptiles
52. Hydrophobia ā Fear of water
IMPORTANT RULES in Medicine
⨠Rule of HALVES ā Hypertension & Diabetes
⢠Only 50% of individuals with hypertension or diabetes are diagnosed.
⢠Of those diagnosed, only 50% receive treatment.
⢠Of those treated, only 50% achieve control of their condition.
⢠This rule highlights the gaps in diagnosis, treatment, and management.
āø»
⨠Rule of 5 ā Pregnancy
⢠This rule is related to the size of the uterus during pregnancy:
⢠5 weeks ā Uterus is just palpable.
⢠10 weeks ā Uterus reaches the pubic symphysis.
⢠15 weeks ā Uterus is midway between the pubic symphysis and umbilicus.
⢠20 weeks ā Uterus reaches the umbilicus.
⢠25 weeks and beyond ā Uterus continues to grow progressively.
āø»
⨠Rule of 9 ā Burns
⢠Used to estimate Total Body Surface Area (TBSA) affected by burns:
⢠Head & Neck ā 9%
⢠Each Arm ā 9% (4.5% front, 4.5% back)
⢠Each Leg ā 18% (9% front, 9% back)
⢠Front of Torso ā 18%
⢠Back of Torso ā 18%
⢠Perineum ā 1%
⢠Helps guide fluid resuscitation and management.
āø»
⨠Rule of 10 ā Cleft Lip & Palate
⢠Used to determine optimal timing for cleft lip repair (Rule of 10s):
⢠10 weeks old
⢠10 pounds (4.5 kg) weight
⢠Hemoglobin of 10 g/dL
⢠Ensures the baby is healthy enough for surgery.
āø»
⨠Rule of 12 ā Rigor Mortis
⢠Refers to the onset and resolution of rigor mortis in a 12-hour cycle:
⢠First 12 hours ā Rigor mortis develops.
⢠Next 12 hours ā Rigor mortis is fully established.
⢠Final 12 hours ā Rigor mortis disappears.
⢠Used in forensic medicine to estimate time of death.
āø»
⨠Rule of 15 ā Hypoglycemia
⢠Used to manage hypoglycemia (low blood sugar levels):
⢠If blood glucose is ā¤70 mg/dL, give 15g of fast-acting carbohydrate (e.g., glucose tablets, juice).
⢠Wait 15 minutes and recheck blood glucose.
⢠If still low, repeat 15g of glucose and recheck after another 15 minutes.
⢠Once stable, eat a small meal or snack to prevent recurrence.
PORTAL HYPERTENSION
Normal portal vein pressure is 5-10 mm of Hg.
Poratl hypertension is defined as an elevation of portalvenous pressure> 10-12 mm of Hg
It is most common cause of Gastroi.
# Infection control
Central line infections can be prevented by following proper hygiene practices and using antiseptics. Patients and their families can also ask their healthcare provider about their options and how to care for the line.
Patient practices :-
* Ask your healthcare provider to clean their hands and wear gloves before touching the catheter
* Avoid touching the tubing or where it exits the skin
* Ask your healthcare provider if you still need the catheter
* Remove the catheter as soon as it's no longer needed
Healthcare provider practices :-
* Use chlorhexidine or povidone iodine to clean the catheter opening
* Use sterile devices to access catheters
* Change dressings regularly, especially if they're wet or soiled
* Use aseptic technique when changing dressings
* Use the minimum number of ports or lumens needed
Other practices :-
* Wash hands before entering and leaving a patient's room
* Follow recommended infection control guidelines
* Use full-barrier precautions when inserting the catheter
* Avoid using the femoral vein for adult patients
ANATOMY LENGTHS
4cm long structures in the body
šInguinal canal
šAnal canal
šFemale urethra
šAuditory tube
šCystic duct
šOptic nerve
šProstatic urethra
šLeft principle bronchus
5cm long structures in the body
šMale larynx
šLeft primary bronchus
šParotid duct
šSubmandibular duct
šLateral lobe of thyroid gland
šTestis
šFirst part of duodenum
šNeck of femur
šLateral wall of bony orbit
šMedial wall of bony orbit
7.5cm long structures in the body
šAnterior wall of vagina
šVirgin uterus
šSecond part of duodenum
šBile duct
9cm long structures in the body
šAppendix
šMultiparous uterus
šPosterior wall of vagina
11cm long structures in the body
šTrachea
šKidney
šUterine tube
šThird part of duodenum
12cm long structures in the body
šRectum
šSpleen
šPharynx
15cm long structures in the body
šAdductor canal
šAscending colon
šPancreas
šRoot of mesentery
25cm long structures in the body
šOesophagus
šDescending colon
šUreter
šDuodenum
45cm long structures in the body
šSpinal cord
šThoracic duct
šTransverse colon
šFemur
šSartorius
šVas deferens
ā
Father of Biology: *Aristotle*
ā
Father of Physics: *Albert Einstein*
ā
Father of Chemistry: *Jabir Bin Hayan*
ā
Father of Statistics: *Ronald Fisher*
ā
Father of Zoology: *Aristotle*
ā
Father of History: *Herodotus*
ā
Father of Microbiology: *Louis Pasteur*
ā
Father of Botany: *Theophrastus*
ā
Father of Algebra: *Muhammad ibn Musa al-Khwarizmi*
ā
Father of Blood groups: *Landsteiner*
ā
Father of Electricity: *Benjamin Franklin*
ā
Father of Trigonometry: *Hipparchus*
ā
Father of Geometry: *Euclid*
ā
Father of Modern Chemistry: *Antoine Lavoisier*
ā
Father of Robotics: *Engelberger*
ā
Father of Electronics: *Michael Faraday*
ā
Father of Internet: *Vinton Cerf*
ā
Father of Economics: *Adam Smith*
ā
Father of Video game: *Ralph Baer*
ā
Father of Architecture: *Louis Henry Sullivan*
ā
Father of Genetics: *Gregor Johann Mendel*
ā
Father of Nanotechnology: *Heinrich Rohrer*
ā
Father of C language: *Dennis Ritchie*
ā
Father of World Wide Web: *Tim Berners-Lee*
ā
Father of Search engine: *Alan Emtage*
ā
Father of Periodic table: *Dmitri Mendeleev*
ā
Father of Taxonomy: *Carolus Linnaeus*
ā
Father of Surgery (early): *Sushruta*
ā
Father of Mathematics: *Archimedes*
ā
Father of Medicine: *Hippocrates*
ā
Father of Homeopathy: *Samuel Hahnemann*...!
*CVP Line*
⢠Preferred site of CVP insertion: *Right internal jugular vein*
⢠M/C site of insertion for TPN administration: *Subclavian vein*
⢠M/C site with least risk of infection: *Subclavian vein*
⢠M/C site with high risk of infection and DVT: *Femoral vein*
⢠M/C site in emergency: *Femoral Vein*
⢠High risk of pneumothorax: *Subclavian*
⢠Patient position while inserting the CVP line through jugular or subclavian vein: *Trendelenburg*
⢠M/C site of insertion of PICC line: *Basilic vein*
⢠Technique used to insert the CVAC: *Seldinger technique*
⢠Solution for cleaning insertion site: *2% CHG*
Nutritional programme
āļøMid-Day Meal Programme -1961
āļøSchool Health Programme - 1962
āļøNational Goiter control programme -1962
āļøApplied Nutrition Programme -1963
āļøVit-A Prophylaxis programme - 1970
āļøSpecial Nutrition Programme -1970
āļøBalwadi Nutrition Programme -1970
āļøMid day meal Scheme -15 Aug 1995
*-Disposable Of PPE*
1. *Head Cover:* Yellow š”
2. *Goggle:* Red š“
3. *Face Shield:* Red š“
4. *Surgical Mask:* Yellow š”
5. *N-95 Mask:* Yellow š”
6. *Surgical Gown:* Yellow š”
7. *Isolation Grown:* Yellow š”
8. *Coverall Suits:* Yellow š”
9. *Gloves:* Red š“
10. *Shoes:* Yellow š”
_š¹TUBERCULOSIS CASE MANAGEMENT_
_š¹DIAGNOSIS OF TB_
āŖļøThe presence of pulmonary tuberculosis should be suspected in individuals presenting with one or more of the following complaints:
āŖļøCough for 1 week or longer,
āŖļøNight sweats
āŖļøFever
āŖļøLoss of weight,
āŖļøA body mass index(BMI) <17kg/m2 or failure to thrive among children.
_š¹LABORATORY INVESTIGATIONS_
āŖļøEvery effort should be made to bacteriologically confirm the diagnosis of TB,with rapid molecular test such as the Xpert MTB/Rif test being the preferred diagnostic test for patients being evaluated for tuberculosis.
āŖļø A specimen should be submitted for all cases being investigated for pulmonary TB as per TB diagnostic algorithm, in order to confirm the diagnosis of Tuberculosis.
_š¹Chest X-RAYS_
āŖļøChest X-ray is recommended as a priority SCREENING test for persons classified as being at high risk for tuberculosis.
āŖļøThe following persons are classified as being at high risk for tuberculosis:
ā«ļøChildren under the age of 5 years.
ā«ļøPeople living with HIV.
ā«ļøThose who are malnourished.
ā«ļøPeople above 60 years of age.
ā«ļøPeople with diabetes mellitus.
ā«ļøPeople who drink alcohol excessively
ā«ļøMiners and ex-miners
ā«ļøPeople in congregate settings.
ā«ļøInmates and correctional facility communities.
ā«ļøHealth care workers
āŖļøNote: In the presence of clinical improvement, it is not necessary to monitor the response of pulmonary TB to treatment by chest x-rays.
_TUBERCULIN TESTING_
āŖļø0.1 mls intradermal
āŖļøExamine induration at 48-72 hours.
āŖļøA positive tuberculin skin test (person with normal immunity: induration > 10 mm, person with defective immunity: induration > 5 mm) may indicate active infection (especially if strongly positive) , previous infection or previous BCG.
āŖļøAbsence of a response does not exclude TB because individuals with HIV may not have sufficient immunity for a positive skin test despite active TB.
āŖļøIf a child under 3 years of age has not had BCG,the Mantoux test may be useful.
_š¹TREATMENT REGIMEN FOR TB_
āŖļøOne treatment regimen is now used in Zimbabwe for the treatment of drug sensitive TB,regardless of the treatment history.
āŖļøThe regimen consists of a combination of the four first line medicines HRZE:
H= isoniazid
R= rifampicin
Z= pyrazinamide
E= ethambutol
āŖļøThese medicines are available as Fixed Dose Combination (FDC).
āŖļøThe intention of these combination tablets is to improve compliance by reducing the number of tablets a patient takes, and to reduce the possibility of medicine resistance developing.
āŖļøThe number of FDC tablets is determined by a weight range of each patient at the start of treatment
āŖļøTreatment is the same for HIV infected people as for non-HIV
_š¹TREATMENT OF DRUG-SENSITIVE TB_
āŖļøAll drug sensitive cases of TB regardless of site, severity or previous treatment history are treated using the regimens summarised below.
_š¹BONE HEALING_
āŖļøBone healing, also known as bone fracture repair,is a complex and dynamic process by which the body restores damaged or fractured bone tissue to its original strength and function.
āŖļøThe process of bone healing involves several stages:
_1.FRACTURE HEMATOMA_
āŖļøWhen a fracture occurs,bleeding creates a hematoma,which surrounds the ends of the fragments.
āŖļøThe hematoma is an extravasated blood that changes from a liquid to a semisolid clot.
āŖļøThis occurs in the initial 72 hours after injury.
_2.GRANULATION TISSUE_
āŖļøDuring this stage,active phagocytosis absorbs the products of local necrosis.
āŖļøThe hematoma converts to granulation tissue.
āŖļøGrannulation tissue(consisting of new blood vessels,fibroblasts and osteoblasts) produces the basis for new bone substance called osteoid during 3 days to 14 days post injury.
_3.CALLUS FORMATION_
āŖļøAs minerals (calcium,phosphorus,and magnesium) and new bone matrix are deposited in the osteoid,an organized network of bone is formed that is woven about the fracture parts.
āŖļøCallus is primarily composed of cartilage,osteoblasts,calcium and phosphorus.
āŖļøIt usually appears by the end of the second week after injury.
āŖļøEvidence of callus formation can be verified by x-ray
_4.OSSIFICATION_
āŖļøOccurs from 3 weeks to 6 months after the fracture and continues until the fracture has healed.
āŖļøCallus ossification is sufficient to prevent movement at the fracture site when the bones are gently stressed.
āŖļøHowever,the fracture is still evident on x-ray.
āŖļøDuring this stage of clinical union,the patient may be allowed limited mobility or the cast may be removed.
_5.CONSOLIDATION_
āŖļøAs callus continues to develop,the distance between bone fragments diminishes and eventually closes.
āŖļøDuring this stage ossification continues.
āŖļøIt can be equated with radiological union,which occurs when there is x-ray evidence of complete bone union.
āŖļøThis phase occur up to 1 year after injury
_6.REMODELING_
āŖļøExcess bone tissue is resorbed in the final stage of bone healing, ane union is complete.
āŖļøGradual return of injured bone to its preinjury structural strength and shape occurs.
āŖļøBone remodels in response to physical loading stress or wolf's law.Initially stress is provided through exercise.
*Amlodipine*
*GROUP:* Antihypertensive
*CLASS:* Calcium Channel Blockers(CCBs)
_š¹MODE OF ACTION_
āŖļøIt inhibits the influx(entry) of calcium ions into vascular smooth muscle cells, leading to relaxation and vasodilation.
āŖļøThis vasodilation decreases peripheral resistance, resulting in a decrease in blood pressure.
_š¹ROLE OF CALCIUM IONS IN HUMAN BODY_
āŖļøCalcium is necessary for muscle contractions to occur throughout the body.
āŖļø This mineral enters muscle cells through various ion channels (L,N,T,P and R),which are tiny pores on the surface of the cell.
āŖļøThis process is essential for normal body functioning.
āŖļøCCB's reduce the amount of calcium that can enter muscle cells in the heart and blood vessel walls via L-type calcium-channels.
āŖļøThis lowers the pressure in the blood vessels and on the heart.
_š¹PRESENTATION_
āŖļøTablets;2,5mg,5mg,10mg
āŖļøCapsules;5mg,10mg
_š¹INDICATIONS_
āŖļøHypertension
āŖļøAngina pectoris
_š¹CONTRAINDICATIONS & CAUTIONS_
āŖļøContraindicated in patients hypersensitive to drug.
āŖļøUse cautiously in patients receiving other peripheral vasodilators, especially those with severe aortic stenosis, and in those with heart failure.
āŖļøBecause drug is metabolized by the liver,use cautiously and in reduced dosage in patients with severe hepatic disease.
_š¹DOSE,ROUTE,FREQUENCY_
āŖļøAdults:5-10 mg per oral once daily as long term.
āŖļøPaeds:2.5mg - 5mg per oral once daily
_š¹HALF-LIFE_
āŖļø30 to 50 hours.
_š¹ADVERSE REACTIONS_
_š¹CENTRAL NERVOUS SYSTEM_
āŖļø Headache.
āŖļøSomnolence.
āŖļøDizziness.
āŖļøLight-headedness.
āŖļøAsthenia.
_š¹CARDIOVASCULAR SYSTEM_
āŖļøOedema.
āŖļøFlushing.
āŖļøPalpitations.
_š¹GASTROINTESTINAL_
āŖļøDyspepsia.
āŖļø Nausea.
āŖļøAbdominal pain.
_š¹GENITOURINARY_
āŖļøSexual difficulties.
_š¹MUSCULOSKELETAL_
āŖļøMuscle cramps.
_š¹RESPIRATORY_
āŖļøDyspnea.
_š¹SKIN_
āŖļøRash.
āŖļøPruritus.
_š¹INTERACTIONS_
āŖļøNone reported.
_š¹EFFECTS ON LAB TEST RESULTS_
āŖļøNone reported.
_š¹OVERDOSE S&S:_
āŖļøHypotension.
_š¹NURSING CONSIDERATIONS_
āŖļøMonitor patient carefully.Some patients,especially those with severe obstructive coronary artery disease,have developed increased frequency,duration,or severity of angina or acute Myocardial infarction after initiation of calcium channel blocker therapy or at time of dosage increase.
āŖļøMonitor blood pressure frequently during initiation of therapy because drug-induced vasodilation has a gradual onset,acute
hypotension is rare.
āŖļøAbrupt withdrawal of drug may increase frequency and duration of chest pain.Taper dose gradually under medical
supervision.
āŖļøLook alikeāsound alike: Donāt confuse amlodipine with amiloride.
_š¹PATIENT TEACHING_
āŖļøCaution patient to continue taking drug,even when he feels better.
āŖļøTell patient that nitroglycerin may be taken as needed when angina symptoms are acute.If patient continues nitrate therapy during adjustment of amlodipine dosage,urge continued compliance.
*_Native valve endocarditis_*
āŖļøBenzylpenicilin 5MU 6 hourly 2- 6 weeks.
āŖļøPaeds dose:0.05-0.1MU/kg
āŖļø Gentamycin 80-120mg IV 12 hourly for 2 weeks.
āŖļøPaeds dose: 1-1.5 mg/kg.
āŖļøCeftriaxone 1g IV 12hrly for 2-6 weeks.
āŖļøPaeds dose:50mg/kg
*_Prosthetic valve endocarditis_*
āŖļøCloxacillin 2g 6hrly for 4-6 weeks.
āŖļøPaeds dose:15mg/kg
āŖļøGentamycin 80-120mg 12hrly for 4 weeks.
āŖļøPaeds dose:1-1.5mg/kg
*_Treatment of culture positive endocarditis_*
āŖļøBenzylpenicilin 5MU IV 6hrly for 4-6 weeks.
āŖļøGentamycin 80-120mg 12hrly for 4 weeks.
_š¹COMPLICATIONS_
āŖļøValvular heart disease.
āŖļø Heart failure.
āŖļøMyocarditis.
_š¹ENDOCARDITIS_
āŖļøEndocarditis is an inflammation of the inner layer of the heart.
āŖļøIt is usually caused by bacteria, fungi, or viruses entering the bloodstream through invasive procedures like dental work or surgery.
āŖļøThe microbes accumulate on the heart valves, forming vegetations that can damage the valves over time.
_š¹CAUSES_
*_(a)Bacteria._*
āŖļøStreptococcus viridan.
āŖļøStaphylococcus Aureus.
_*(b)Fungi*_
āŖļøAspagellus
āŖļøCandida
*_(c).Acute rheumatic fever which causes enlarged and tender lymph nodes,damages the valves._*
_*(d).Congenital heart disease.*_
_š¹RISK FACTORS_
āŖļøPrevious heart damage.
āŖļøHeart surgeries.
āŖļøDental surgeries can introduce bacterias.
āŖļøLong term haemodialysis.
āŖļøDiabetes Mellitus.
*_š¹TYPES_*
_*(I)ACUTE ENDOCARDITIS*_
āŖļøInfections caused by staphylococcus Aureus occurs rapidly on normal valves.
_*(II)SUBACUTE ENDOCARDITIS*_
āŖļøThis is infection caused by streptococcus viridans and usually develops gradual on previously damaged heart valves e.g. rheumatic heart disease.
*_(III)POST OPERATIVE ENDOCARDITIS_*
āŖļø Infection following cardiac surgery in which prosthetic heart valves are used.
āŖļø It is caused by staphylococcus.
*_š¹PATHOPHYSIOLOGY_*
āŖļøA deformity or injury of the endocardium leads to accumulation fibrin and platelets (clot
formation).
āŖļøThe infection most frequently results in
platelets,fibrin,blood cells,and microorganisms that clusteras vegetations on the endocardium.
āŖļøThe vegetations may embolize to other tissues throughout the body.
āŖļøAs the clot on the endocardium continues to expand,the infecting organism is covered by the new clot and concealed from the body's normal defenses.
āŖļøThe infection may erode through the endocardium into the underlying structures(eg,valve
leaflets),causing tears or other deformities of valve leaflets,dehiscence of prosthetic valves,
deformity of the chordae tendineae,or mural abscesses.
āŖļøUsually the onset of infective endocarditis is insidious.
āŖļøThe signs and symptoms develop from the toxic effect of the infection,from destruction of the
heart valves,and from embolization of fragments of vegetative growths on the heart.
āŖļøSystemic emboli occur with lef-sided heart infective endocardis;pulmonary emboli occur with
right-sided heart infective endocarditis.
_OR_
āŖļøThe endocardium is invaded by microbes directly.
āŖļøThe microbes cause deformity, thickening, stiffening and scaring of the valve leaflets and may travel to other organs and obstruct blood flow.
āŖļø This results in valvular incompetence and invasion of the myocardium by infection resulting myocardial dysfunction, sepsis and congestive cardiac failure.
_š¹CLINICAL FEATURES_
āŖļøFever.
āŖļøTachycardia
āŖļøPetechiae conjunctiva.
āŖļøSplinter hemorrhages in nail beds.
āŖļøOsler's nodes -painful red nodes on pads of fingers and toes; usually late sign of infection and found with a sub acute infection.
āŖļøMurmurs with other signs and symptoms may indicate right side heart infection.
āŖļøTachycardia related to decreased cardiac output.
āŖļøLocalized headaches.
āŖļøTransient cerebral ischemia.
āŖļø Altered mental status, aphasia.
āŖļøHemiplegia.
āŖļø Cortical sensory loss
āŖļøHaemoptysis.
āŖļøChest pain, shortness of breath.
āŖļøKidney --haematuria.
āŖļøSpleen- pain in left upper quadrant of abdomen radiating to left shoulder.
_š¹DIAGNOSTIC EVALUATIONS_
āŖļøBlood cultures for isolation of the causative organism.
āŖļøEchocardiogram for identification of infected area.
āŖļø Blood for white cell count and erythrocyte sedimentation rate which show elevated levels.
āŖļøCXR to detect cardiomegaly.
āŖļøUrinalysis to see microscopic hematuria.
āŖļøComputerized tomography rule out heart damage
āŖļøReview bun and creatinine levels to evaluate renal function.
āŖļøReview endocardiography finding if available for valvuler and
ventricular function and presence of vegetation.
_š¹MANAGEMENT_
Here are some common diseases related to the digestive system:
1. Gastroesophageal Reflux Disease (GERD)
2. Inflammatory Bowel Disease (IBD)
- Crohn's disease
- Ulcerative colitis
3. Irritable Bowel Syndrome (IBS)
4. Peptic Ulcer Disease
5. Diverticulitis
6. Gastrointestinal Bleeding
7. Celiac Disease
8. Chronic Pancreatitis
9. Gallstones
10. Cholecystitis (inflammation of the gallbladder)
11. Hepatitis (liver inflammation)
12. Cirrhosis (liver scarring)
13. Liver Cancer
14. Gastrointestinal Stromal Tumors (GISTs)
15. Colon Cancer
16. Rectal Cancer
17. Anal Cancer
18. Dysphagia (swallowing difficulty)
19. Achalasia (esophageal motility disorder)
20. Scleroderma (systemic sclerosis)
These diseases can be categorized into various groups, such as:
- Inflammatory conditions (e.g., IBD, celiac disease)
- Functional disorders (e.g., IBS, dyspepsia)
- Structural disorders (e.g., peptic ulcer disease, diverticulitis)
- Neoplastic disorders (e.g., colon cancer, GISTs)
- Infections (e.g., hepatitis, gastroenteritis)
- Motility disorders (e.g., achalasia, scleroderma)
It's important to note that this is not an exhaustive list, and there are many other diseases and conditions that can affect the digestive system.
*Actinic Keratosis (AK)*
- A precancerous skin condition caused by prolonged exposure to UV radiation from the sun or tanning beds
- Characterized by rough, scaly, or crusty patches on sun-exposed areas (face, ears, hands, arms, and legs)
- Can progress to squamous cell carcinoma (SCC) if left untreated
- Typically affects fair-skinned individuals over 40 years old
- Treatment options include:
- Topical creams or gels (e.g., fluorouracil, imiquimod)
- Cryotherapy (freezing)
- Photodynamic therapy (PDT)
- Surgical excision
- Prevention: sun protection (sunscreen, clothing, hats), avoid tanning beds, and regular skin checks
Early detection and treatment can prevent progression to SCC and reduce the risk of complications.
ā” Mirena is a hormonal intrauterine device (IUD) that provides long-term contraception and is approved for the treatment of heavy menstrual bleeding (menorrhagia). It releases a small amount of the hormone levonorgestrel, which helps reduce menstrual flow and prevent pregnancy for up to 5-8 years.
ā”Minipill : A progestin-only oral contraceptive, not an IUD.
ā” Copper T : A non-hormonal IUD that provides long-term contraception but does not treat menorrhagia and may worsen bleeding.
ā” Norplant (Subdermal) : A set of hormone-releasing implants placed under the skin for contraception but not specifically used to treat menorrhagia.
Hyperphosphatemia (elevated levels of phosphate in the blood) significantly affects the heart, especially in individuals with chronic kidney disease (CKD) or other metabolic disorders. Hereās the pathophysiology:
1. Vascular Calcification
⢠High phosphate levels lead to vascular smooth muscle cell (VSMC) transformation into osteoblast-like cells.
⢠This triggers deposition of calcium-phosphate in the arterial walls, resulting in vascular calcification.
⢠Calcification reduces the flexibility of blood vessels, increasing vascular stiffness and contributing to hypertension and increased afterload on the heart.
2. Cardiac Calcification
⢠Hyperphosphatemia can also lead to calcification of the heart valves (e.g., aortic and mitral valves) and myocardium.
⢠Valve calcification can impair their function, causing stenosis or regurgitation, which increases the workload of the heart and predisposes to heart failure.
3. Endothelial Dysfunction
⢠Elevated phosphate disrupts endothelial cell function, reducing nitric oxide (NO) production and promoting oxidative stress.
⢠This contributes to arterial stiffness, increased vascular resistance, and heightened risk of atherosclerosis.
4. Left Ventricular Hypertrophy (LVH)
⢠Increased vascular resistance and calcification elevate cardiac workload, causing left ventricular hypertrophy.
⢠LVH is associated with poor cardiac output and increased risk of arrhythmias, heart failure, and sudden cardiac death.
5. Secondary Hyperparathyroidism
⢠In CKD, hyperphosphatemia often coexists with secondary hyperparathyroidism due to disrupted calcium-phosphate balance.
⢠Elevated parathyroid hormone (PTH) levels further promote bone resorption and calcium-phosphate deposition in tissues, worsening vascular calcification and cardiac dysfunction.
6. Pro-Inflammatory State
⢠Hyperphosphatemia induces systemic inflammation, which exacerbates atherosclerosis and impairs cardiac function.
Clinical Implications
Hyperphosphatemia is a major risk factor for cardiovascular morbidity and mortality, especially in CKD patients. Managing phosphate levels through dietary restrictions, phosphate binders, and maintaining calcium-phosphate balance is crucial to prevent or mitigate these cardiac complications.
MEDICAL TERMINOLOGIES
1. Spinal fusion - Surgical joining of vertebrae
2. Splenomegaly - Enlarged spleen
3. Spondylitis - Inflammation of spine
4. Spondylosis - Degeneration of spine
5. Squamous cell carcinoma - Skin cancer
6. Stenosis - Narrowing of blood vessel
7. Sterilization - Germ killing
8. Stethoscope - Heart and lung examination device
9. Stomatitis - Mouth inflammation
10. Stroke - Brain attack
11. Subarachnoid hemorrhage - Bleeding in brain
12. Subcutaneous - Under the skin
13. Sudden infant death syndrome (SIDS) - Unexplained infant death
14. Suicide - Intentional self-harm
15. Sulfonamide - Antibacterial medication
16. Superficial - Near the surface
17. Supination - Rotating upward
18.Supraventricular tachycardia - Fast heartbeat
19. Surgery - Operation
20. Suture - Stitch
21. Sympathectomy - Nerve removal
22. Symptom - Disease sign
23. Syncope - Fainting
24. Syndactyly - Fused fingers or toes
25. Syndrome - Group of symptoms
26. Synovitis - Joint inflammation
27. Systemic lupus erythematosus (SLE) - Autoimmune disorder
28. Tachycardia - Fast heartbeat
29. Tachypnea - Rapid breathing
30. Talipes equinovarus (TEV) - Clubfoot
31. Tamoxifen - Breast cancer medication
32. Tardive dyskinesia - Movement disorder
33. Tarsal tunnel syndrome - Foot nerve compression
34. Tendonitis - Tendon inflammation
35. Tenosynovitis - Tendon sheath inflammation
36. Testicular torsion - Twisted testicle
37. Thalassemia - Blood disorder
38. Thallium - Heavy metal poisoning
39. Thoracentesis - Chest fluid removal
40. Thrombocytopenia - Low platelet count
41. Thrombolysis - Blood clot dissolution
42. Thrombophlebitis - Vein inflammation
43. Thrombosis - Blood clot
44. Thyroid crisis - Thyroid storm
45. Thyroiditis - Thyroid inflammation
46. Tinnitus - Ringing in ears
47. Tonsillitis - Tonsil inflammation
48. Topical - Applied to skin
49. Torsion - Twisting
50. Torticollis - Twisted neck
51. Total parenteral nutrition (TPN) - Intravenous nutrition
52. Toxemia - Poisoning
53. Toxoplasmosis - Parasitic infection
54. Tracheitis - Windpipe inflammation
55. Tracheostomy - Breathing tube
56. Trachoma - Eye infection
57. Transfusion - Blood transfer
58. Transient ischemic attack (TIA) - Mini-stroke
59. Transplant - Organ replacement
60. Trauma - Injury
61. Tremor - Shaking
62. Trichomoniasis - Vaginal infection
63. Triglyceride - Blood fat
64. Triiodothyronine (T3) - Thyroid hormone
65. Troponin - Heart attack marker
66. Tuberculosis (TB) - Infection
67. Tumor - Abnormal growth
68. Tympanic membrane - Eardrum
69. Tympanitis - Middle ear inflammation
70. Tyrosinemia - Genetic disorder
71. Ulcer - Wound
72. Ultrasound - Imaging test
73. Unconscious - Comatose
74. Ureters - Kidney tubes
75. Urethra - Urinary tube
76. Uric acid - Waste product
77. Urinalysis - Urine examination
78. Urology - Urinary specialty
79. Uterine fibroids - Tumors in uterus
80. Uveitis - Eye inflammation
81. Vagina - Female genital canal
82. Vaginitis - Vaginal inflammation
83. Vagotomy - Vagus nerve cutting
84. Valvuloplasty - Heart valve repair
85. Varicella - Chickenpox
86. Varicocele - Enlarged veins in scrotum
87. Vasculitis - Blood vessel inflammation
88. Vasodilation - Blood vessel widening
89. Vasopressor - Blood pressure medication
90. Venereal disease - Sexually transmitted disease
Lists of Phobiaās
1. Achluophobia ā Fear of darkness
2. Acrophobia ā Fear of heights
3. Aerophobia ā Fear of flying
4. Algophobia ā Fear of pain
5. Agoraphobia ā Fear of open spaces or crowds
6. Aichimophobia ā Fear of pointed objects
7. Amaxophobia ā Fear of riding in a car
8. Androphobia ā Fear of men
9. Anginophobia ā Fear of angina or choking
10. Anthrophobia ā Fear of flowers
11. Anthropophobia ā Fear of people or society
12. Aphenphosmphobia ā Fear of being touched.
13. Arithmophobia ā Fear of numbers
14. Astraphobia ā Fear of thunder and lightning
15. Ataxophobia ā Fear of disorder or untidiness
16. Atelophobia ā Fear of imperfection
17. Atychiphobia ā Fear of failure
18. Autophobia ā Fear of being alone
19. Bacteriophobia ā Fear of bacteria
20. Barophobia ā Fear of gravity21. Bathmophobia ā Fear of stairs and steep slopes
22. Batrachophobia ā Fear of amphibians
23. Belonephobia ā Fear of pins and needles
24. Bibliophobia ā Fear of books
25. Botanophobia ā Fear of plants
26. Cacophobia ā Fear of ugliness
27. Catagelophobia ā Fear of being ridiculed
28. Catoptrophobia ā Fear of mirrors
29. Chionophobia ā Fear of snow
30. Chromophobia ā Fear of colors
31. Chronomentrophobia ā Fear of clocks
32. Claustrophobia ā Fear of confined places
33. Coulrophobia ā Fear of clowns
34. Cyberphobia ā Fear of computers
35. Cynophobia ā Fear of dogs
36. Dendrophobia ā Fear of trees
37. Dentophobia ā Fear of dentists
38. Domatophobia ā Fear of houses
39. Dystychiphobia ā Fear of accidents
40. Ecophobia ā Fear of the home
41. Elurophobia ā Fear of cats42. Entomphobia ā Fear of insects
43. Ephebiphobia ā Fear of teenagers
44. Equinophobia ā Fear of horses
45. Gamophobia ā Fear of marriage
46. Genuphobia ā Fear of knees
47. Glossophobia ā Fear of speaking in public
48. Gynophobia ā Fear of women
49. Heliophobia ā Fear of the sun
50. Hemophobia ā Fear of blood
51. Herpetophobia ā Fear of reptiles
52. Hydrophobia ā Fear of water
