Lina Sudanses local Derma Telegram

Most abundant collagen in fetus is A. Collagen 1 B. Collagen 3 C. Collagen 7 D. Collagen 4

■1415 Local derma part 1 2022 Local derma part 1 2019

retinol (retinoid), a topical form of vitamin A, can help treat and prevent inflammatory  acne  lesions.

Explanation

Retinoid

Answer

Nodulocystic acne is treated by A. systemic steroids B. Antibiotic C. Retinoid

■1414 Local derma part 1 2022 Local derma part 1 2018

The late secondary syphilis lesions are nodular and ulcerative. They are only distinguished from the tertiary form by the time of onset and by the presence of spirochetes in the serum. Lesions are commonly in the shape of rings or semi-rings. Patchy hair loss on the scalp may occur.

■■The rash may be elevated, discolored, pus-producing, or eroded. Flat, moist, and warty lesions in the groin and axillae are called condylomata lata. They are usually flooded with spirochetes.

Secondary syphilis presents as a rash characteristically on the palms of the hands and soles of the feet. Additionally, the entire body may be involved including the mouth and genitalia.

■■■The most common sign of secondary syphilis is a rash that does not itch or cause pain. The rash may appear on one part of the body or be more widespread.

Syphilis is a sexually transmitted infection caused by Treponema pallidum, a spirochete. Syphilis is known as the "great imitator” because of its varying clinical signs and symptoms. The infection is characterized by episodes of active disease (primary, secondary and tertiary) with latent periods in between. Syphilis is contagious and transmitted in three ways: (1) intimate contact with an infectious lesion (most common) (2) blood transfusion (3) during delivery of a baby he/she may contract syphilis from the birth can

Explanation

Condyloma lata

Answer

In secondary syphilis charecteristic sign to look for is: A. Itchy papules B. codyloma lata

■1413 Also review Question no 724

IL-16 promotes eosinophil migration in vitro, by activating the plasminogen-plasmin system and increasing the membrane expression of its receptor. This effect is initiated via CD4 and mediated via the release of CCR3 ligand chemokines. 

Explanation