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الجماعة مسلميها من اول كيس
مو كاتبتلكم الدكتورة بين قوسين بالمحاضرة (Nitrofurantion not use in third trimester)
A 24-year-old primigravida at 18 weeks gestation attends a routine antenatal visit. She feels well and has no urinary symptoms. Routine urine culture shows >100,000 CFU/mL of E. coli. Physical examination is normal and she is afebrile. The obstetrician diagnoses asymptomatic bacteriuria in pregnancy.
Which of the following is the most appropriate treatment?
A. Oral Nitrofurantoin for 3 days
B. Oral Nitrofurantoin for 7 days
C. iV Nitrofurantoin
D. IV cephalxin
E. oral cephalxin for 7 days
A 25-year-old pregnant woman at 30 weeks gestation presents with high-grade fever, chills, right flank pain, nausea, and dysuria for 2 days. On examination, her temperature is 39°C and she has marked right costo-vertebral angle tenderness. Urinalysis shows numerous white blood cells and positive nitrites. The obstetrician diagnoses acute pyelonephritis in pregnancy.
Which of the following is the most appropriate antibiotic treatment?
A. IV Nitrofurantoin
B. IV Ciprofloxacin
C. IV Ceftriaxone
D. IV Tetracycline
E. IV Trimethoprim
A 27-year-old pregnant woman at 28 weeks gestation has had three episodes of urinary tract infection during this pregnancy. She is currently asymptomatic after completing treatment for her last UTI. Her obstetrician decides to start prophylactic therapy to prevent recurrent infections during the remainder of pregnancy.
Which of the following is the best antibiotic for this patient ?
A. Ciprofloxacin
B. Amoxicillin
C. Tetracycline
D. Chloramphenicol
E. Trimethoprim-sulfamethoxazole
A 30-year-old pregnant woman at 36 weeks gestation presents with burning micturition, urinary frequency, and lower abdominal discomfort for 2 days. She has no fever or loin pain. Urinalysis shows positive leukocytes and nitrites, consistent with urinary tract infection.
Which of the following is the best antibiotic choice for this patient ?
A. Tetracycline
B. Nitrofurantion
C. Trimethoprim
D. Cephalexin
E. Gentamicin
راح ادز 4 كيسات عن
Mangment of urinary disorder in pregnancy
و ورما ادزهن اربعتهن ادز التوضيح
الي يجاوبهن كلهن صح اولاً السبت يجيب امتياز ثانياً يضمنله امسكيو بالامتحان ثالثاً خليفتح عيادة نسائية على حسابي واني افتحله مجمع طبي رابعاً ينطيني رقم العيادة علمود مستقبلاً زوجتي تراجع يمه
الي بعده ماقاري خليحولهن سيڤ مسج ويرجعلهن وره ميقره المحاضره*
ذني الحالات الي تستوجب بيها انو نكمل ال external fetal monitoring (EFM)
بالمد جابت كيس المختصر مالته طفل عنده فريش وبرايت ميوسين شنو البيست نيكست ستيب اوف مانجمنت ؟ continue EFM
A 24-year-old primigravida at 40 weeks is in active labour. She is receiving oxytocin augmentation.
CTG shows:
Baseline fetal heart rate: 170 bpm
Reduced variability: <5 bpm
Recurrent late decelerations
Initial conservative management was done:
Left lateral position
Oxygen given
IV fluids started
Oxytocin stopped
After 20 minutes, the CTG remains suspicious with no improvement.
On vaginal examination:
Cervix = 5 cm dilated
Membranes ruptured
No contraindication to scalp sampling
What is the best next step in management?
A. Continue labour with reassurance
B. Immediate cesarean section
C. Fetal blood sampling
D. Instrumental delivery
E. Restart oxytocin
بمحاضرة ال mangment of labour
ال frequency of contractions وال heart rate
ينقاسن كل 30 دقيقة
ال pulse rate
ينقاس كل ساعه
ال veginal examination و ال BP وال Temperature
ينقاسن كل 4 ساعات
Never call it malpresentation before 36 weeks because frequently changing position
🔑 Exam principle :-
• Only after 36 weeks does malpresentation become clinically significant (because spontaneous version becomes unlikely)
☑️ This is why , External cephalic version (ECV) manoeuvre is offered at 36–37 weeks, not earlier
📕 Hyperemesis gravidarum ،
the first step of Management is volume resuscitation by 👉 0.9% Normal Saline or Ringer’s lactate , NOT Glucose Water
Hyperemesis gravidarum → severe malnutrition
Persistent vomiting → ↓ oral intake
Loss of vitamins → especially
thiamine depletion
📍 Core idea :-
Giving glucose ( dextrose ) before correcting thiamine (vitamin B1) deficiency can precipitate
acute Wernicke encephalopathy
Because Thiamine is essential for glucose metabolism
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