Radiology Unplugged Dr Sumer Sethi
الذهاب إلى القناة على Telegram
Dr Sumer Sethi this channel to interact with medical students & residents about Radiology and will discuss cases and quizzes here. Should make a difference in your radiology understanding and score in NEETPG INICET FMGE
إظهار المزيد📈 نظرة تحليلية على قناة تيليجرام Radiology Unplugged Dr Sumer Sethi
تُعد قناة Radiology Unplugged Dr Sumer Sethi (@radiologywithsumer) في القطاع اللغوي الإنكليزية لاعباً نشطاً. يضم المجتمع حالياً 33 682 مشتركاً، محتلاً المرتبة 567 في فئة الطب والمرتبة 12 031 في منطقة الهند.
📊 مؤشرات الجمهور والحراك
منذ تأسيسه في невідомо، حقق المشروع نمواً سريعاً وجمع 33 682 مشتركاً.
بحسب آخر البيانات بتاريخ 08 يوليو, 2026، تحافظ القناة على نشاط مستقر. خلال آخر 30 يوماً تغيّر عدد الأعضاء بمقدار 210، وفي آخر 24 ساعة بمقدار 24، مع بقاء الوصول العام مرتفعاً.
- حالة التحقق: غير موثّقة
- معدل التفاعل (ER): يبلغ متوسط تفاعل الجمهور 7.87%. وخلال أول 24 ساعة من النشر يحصد المحتوى عادةً 4.37% من ردود الفعل نسبةً إلى إجمالي المشتركين.
- وصول المنشورات: يحصل كل منشور على متوسط 2 643 مشاهدة. وخلال اليوم الأول يجمع عادةً 1 468 مشاهدة.
- التفاعلات والاستجابة: يتفاعل الجمهور بانتظام؛ متوسط التفاعلات لكل منشور يبلغ 3.
- الاهتمامات الموضوعية: يركز المحتوى على مواضيع رئيسية مثل dam, alpha, climb, recognition, custom.
📝 الوصف وسياسة المحتوى
يصف المؤلف القناة بأنها مساحة للتعبير عن الآراء الذاتية:
“Dr Sumer Sethi this channel to interact with medical students & residents about Radiology and will discuss cases and quizzes here. Should make a difference in your radiology understanding and score in NEETPG INICET FMGE”
بفضل وتيرة التحديث المرتفعة (أحدث البيانات بتاريخ 09 يوليو, 2026) تحافظ القناة على حداثتها ومستوى وصول مرتفع. وتُظهر التحليلات تفاعلاً نشطاً من الجمهور، ما يجعلها نقطة تأثير مهمة ضمن فئة الطب.
33 682
المشتركون
+2424 ساعات
+1577 أيام
+21030 أيام
جاري تحميل البيانات...
القنوات المماثلة
سحابة العلامات
الإشارات الواردة والصادرة
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جذب المشتركين
يوليو '26
يوليو '26
+173
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يونيو '26
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مايو '26
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أبريل '26
+178
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مارس '26
+562
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فبراير '26
+45
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يناير '26
+430
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ديسمبر '25
+155
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+159
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+499
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أغسطس '25
+264
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يوليو '25
+1 127
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مايو '25
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أبريل '25
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مارس '25
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فبراير '25
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يناير '25
+674
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ديسمبر '24
+1 323
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نوفمبر '24
+952
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أكتوبر '24
+911
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سبتمبر '24
+1 927
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أغسطس '24
+1 358
في 2 قنوات
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يوليو '24
+2 378
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يونيو '24
+2 064
في 2 قنوات
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مايو '24
+2 728
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أبريل '24
+1 022
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مارس '24
+1 165
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فبراير '24
+10 382
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| التاريخ | نمو المشتركين | الإشارات | القنوات | |
| 08 يوليو | +25 | |||
| 07 يوليو | +16 | |||
| 06 يوليو | +87 | |||
| 05 يوليو | 0 | |||
| 04 يوليو | +4 | |||
| 03 يوليو | +14 | |||
| 02 يوليو | +23 | |||
| 01 يوليو | +4 |
منشورات القناة
Bilateral or multiple angiomyolipomas(AMLs) strongly suggest Tuberous Sclerosis Complex (TSC). Up to 80% of patients with bilateral AMLs have TSC, an inherited disorder caused by TSC1/TSC2 mutations. Lipid-poor AMLs contain microscopic fat, producing signal drop on opposed-phase MRI despite the absence of macroscopic fat on CT or T1-weighted MRI.
| 2 | A 30-year-old patient has multiple bilateral renal pathology as shown. Which diagnosis is most likely? | 1 440 |
| 3 |  لا يوجد نص... | 1 324 |
| 4 | Pulmonary arterial hypertension (PAH) is characterized by pruning of peripheral pulmonary vessels on chest imaging due to loss of distal pulmonary arteries. The most common genetic cause is a BMPR2 mutation. Hemodynamically, PAH is a precapillary pulmonary hypertension with a pulmonary capillary wedge pressure (PCWP) ≤15 mmHg, which helps exclude Group 2 pulmonary hypertension caused by left heart disease (PCWP >15 mmHg). On examination, patients typically have a loud P2 and a systolic ejection murmur at the left upper sternal border due to increased flow across the pulmonary valve. | 1 340 |
| 5 | An infant presents with progressive dyspnea, poor feeding, failure to thrive, loud P2 with systolic ejection murmur at left upper sternal border. Echo: pulmonary HTN with preserved LVEF. Which combination is most consistent with this condition? | 1 293 |
| 6 | DiGeorge syndrome is caused by a 22q11.2 microdeletion, best confirmed by FISH (or chromosomal microarray), as the deletion is too small for routine karyotyping. Developmental failure of the 3rd and 4th pharyngeal pouches leads to thymic aplasia (causing T-cell deficiency) and parathyroid hypoplasia (causing hypocalcemia with tetany or seizures). Chest X-ray typically shows an absent thymic shadow, while congenital conotruncal cardiac defects. (e.g., truncus arteriosus, tetralogy of Fallot, interrupted aortic arch) are common. | 1 269 |
| 7 | A newborn with severe congenital heart disease and recurrent neonatal infections with shown radiology finding. Which test best confirms the underlying genetic defect? | 1 246 |
| 8 |  لا يوجد نص... | 1 207 |
| 9 | The image and clinical presentation are consistent with Idiopathic Pulmonary Fibrosis (UIP pattern). The initiating event is repetitive alveolar epithelial injury, causing Type II pneumocyte dysfunction and hyperplasia. These injured Type II pneumocytes release profibrotic cytokines (especially TGF-β), which activate fibroblasts and lead to excessive collagen deposition and honeycomb fibrosis. | 1 282 |
| 10 | A 68-year-old man has progressive exertional dyspnea and dry cough. Radiology finding is shown. What is the primary initiating cellular event in process leading to the given condition? | 1 387 |
| 11 |  لا يوجد نص... | 1 365 |
| 12 | Dear NEET PG aspirants,
The biggest learning objective from the recent FMG exam is very clear, your core topics have to be strong and they have to be done in depth.
Exams are moving towards testing the concept behind the fact. Do not depend only on PYQs. Even when you are doing PYTs, remember the topic behind that question needs to be understood thoroughly because the same concept can be tested in a completely new way.
Video-based questions are becoming important. FMG had around 20–25 video-based questions, but the learning is that videos themselves are not going to be extremely difficult. They will mostly come from common, high-yield topics, but the challenge will be integration.
Only a few had direct one-line stems; most were combined with longer clinical scenarios where you had to:
● Identify the keywords
● Pick the relevant finding
● Connect the concepts
● Reach the diagnosis or next step
And this skill comes only with practice.
That is why TNDs, new pattern mocks and GTs become extremely important. Don’t just give tests — analyze them, identify your weak areas and work on them.
For those whose marks have become stagnant, July should be about:
1. Strong revision with LRR
2. Regular testing
3. Analysis of mistakes
4. Strengthening weak topics
In the last phase, DVT becomes extremely important. Ensure you complete your revisions because repeated revision helps you recognize concepts faster, identify keywords in long stems and integrate information quickly.
Also remember, images remain extremely high yield. Every important image covered in DVT should be mastered because the same image-based concepts can easily be converted into video-based questions.
The exam pattern may evolve, but the foundation remains the same:
Strong concepts + repeated revision + test practice + integration skills.
Focus on these, and you will be ready for any pattern.
Dr Deepti | 2 124 |
| 13 | For any DAMS Online and Offline Courses related Inquiry feel free to fill up the form given and our Trained Counsellors team will revert : https://www.damsdelhi.com/admissionform | 2 121 |
| 14 | https://youtube.com/shorts/moKZB8SNOzc?si=-B1kLfMrao-8deUT | 808 |
| 15 | In acute exudative-phase ARDS, high FDG-PET uptake in lungs is primarily due to: | 2 166 |
| 16 |  لا يوجد نص... | 2 035 |
| 17 | A preterm neonate develops respiratory distress soon after birth and requires intubation. Chest X-ray is shown. What is the most likely diagnosis? | 2 019 |
| 18 |  لا يوجد نص... | 1 871 |
| 19 | Inherited or acquired prothrombotic disorders, including antiphospholipid antibody syndrome, Factor V Leiden mutation, and polycythemia vera. Heparin-induced thrombocytopenia (HIT), which can occur after recent heparin exposure, is also a well-recognized cause of thrombosis despite thrombocytopenia. In contrast, von Willebrand disease is a bleeding disorder caused by defective or deficient von Willebrand factor and does not predispose to venous thrombosis. | 1 844 |
| 20 | Which of the following is NOT a recognized predisposing risk factor for condition associated with given finding? | 1 824 |
