FLASH 2nd

اللي مش عليكم فالتحديدات متشغلوش دماغكم بيه

ركزوا عال nucleus فالأناتومي يدكاتره جاتلنا السنة اللي فاتت عملي - Mention 3 nucleus that have a sensory function in medulla مش فاكر الصيغة بس فهمتوا الفكرة ممكن يجيلكم المرة دي motor - كذلك جابت ال cerebellum وكانت عاوزه ال posterolateral fissure وال arterial supply - lesions in any brain artery مهم

فلاش تتمني لكم صوما مقبولا وإفطارا شهيا

👀The Underlying structure ? 👾The pyramid 》pyramidal corticospinal tract. 👾The olive      》 inferior olivary nucleus 👾Gracile         》 gracile nucleus 👾Cuneate      》 cuneate nucleus 👾Tuberculum cinerum      》spinal trigeminal nucleus 👾Medial Eminence    》abducent nucleus 👾Facial Colliculus   》abducent nucleus surrounded by motor facial fibers

ملخص ال brain lobes اللي هتحتاجه فعملي الأناتومي

كل عام وأنتم بخير جعله الله شهر خير وبركة عليكم وجعلكم فيه من عتقائه من النار ❤️✨

الحمد لله كان فيه تبرعات هي كانت مبلغ بسيط بس الحمد لله وصل والنهارده تم اطعام من خلاله اخواتنا في معسكر جباليا بالتحديد في شمال غزه وان شاء الله مكملين على خير❤️ اتمنى الناس اللي كانت بلقانه ان الفلوس توصل تكون اتيقنت دلوقتي من المصداقيه واتمنى ان كلنا نساعد ولو بمبلغ بسيط من المال كله بيوصل بفضل الله❤️ 01017708748 ده رقم فودافون كاش وابعتلي صورة الوصل الخاص بالدفع على @MOHAMEDESMAIEL ربنا يجعله في ميزان حسناتنا يا رب🤲❤️ شير عشان كله يتبرع❤️

علي حسب كام د/رشا، ال spinotectal بتعمل cross in spinal cord

الرجالة اللي كانت بتذاكر ال motor علي اساس انه مش هيتلغي

حل امتحان الفسيو بتاع النهاردة 🔥

الإجابة عشان الpain دا بيتنقل عن طريق الunmyelinated C fibers و النوع دا بيتحمل الpressure انما الfine touch , kinesthetic sensation and vibration sense دول بيتنقلو عن طريق الA-beta fibers و ديه مش بتتحمل الpressure و طبعا فآخر المرض هيحصل loss of pain sensation برضو بس بيتأخر شوي عشان تحمل الunmyelinated C fibers ديه ■There is loss of Kinesthetic sensations leads to sensory ataxia(دكتور سند مأكد عليها) ■Mechanisms that maintain equilibruim are Cerebellum , Eye , Kinesthetic sensations و بتحتاج إتنين منهم عالأقل مش هتعرف تتوازن بواحدة بس منهم عشان كدا مثلا لو واحد عنده loss of kinesthetic sensations هو كدا فاقد واحدة منهم لو قولتله يقفل عينيه كدا هيبقى كأنه فقد التانية فا هيقع علطول و دا إسمه Positive Romberg's sign و دا اللى بيأكدلك إن المريض عنده Sensory ataxia ■المريض بيبقى عنده حاجة برضو اسمها Stamping gait لأنه عشان هو مش حاسس بحركة رجله فا بيرفع رجله جامد و بيضرب بيها جامد فالأرض عشان يحس مكان رجله فين بالظبط ●Peripheral neuritis ■Hypothesia and anathesia in distal parts of limps--->Glove and stock(دكتور سند مأكد على ديه) يبقى كدا ■Jacket destribution--->Syringomyelia ■Glove and Stock--->Peripheral neuritis فى النهاية.. متنساش تصلى عالنبى و تدعى لإخواننا فى فلسطين ربنا ينصرهم على أعداؤهم و يصبر قلوب أهالى الشهداء و ربنا يتقبل شهداءهم برحمته فى جنات الفردوس بإذن الله اللهم آمين❤🇵🇸

Ahmed Wagih: ...Notes in Sensory CNS... -Eye responds to : ■Light--->Vision[Adequate] ■Strong box hit or strong blow---> flashes and stars--->Vision[Greater than adequate] -The terminal nerve endings in pacinian corpuscles is unmyelinated and called --->Receptor or Transducer area -The 1st node of Ranvier is called ---> Spike generating region as it's first area where action potential occur -Local anethesia produce effect 1st node of Ranvier not on the receptor so it blocks Action potential but not block receptor potential -Sensory coding : ■Modality discrimination--->Muller's law and labeled line principle ■Site discrimination--->Law of projection ■Intensity discrimination--->Weber-Fechner law -Protopathic sensations are perceived by Thalamus while Epicritic sensations are perceived by Cerebral cortex *But final termination of both is Sensory cortex* ●Pain sensation: -Pain is non specific sensation as it can be caused by chemical,mechanical,thermal stimuli -Pain has specific receptors(Free nerve endings) -Pain receptors don't obey muller's law as most of them are polymodal -Pain receptors are : ■Abundant in skin and some internal organs ■Few in deep tissues and all viscera *Very Important*■Brain itself and Parenchymal tissues like: liver , lung , kidney and spleen have no pain receptors--->Pain insensitive structures(الدكتور مأكد عليها) -Sensitivity of receptors is inversely proportional to its threshold -Chemical transmitter on Fast pain--->Glutamate -Chemical transmitter on Slow pain--->Substance P -A delta fibers blocked by--->Pressure and Hypoxia -Unmyelinated C fibers blocked by--->Local anethesia -Difference in pain senation between different people or individuals not according to pain threshold but according to--->Body reactions to pain -Somatic reflex in body reactions to different types of pain is prepotent reflex--->prevails over other reflexes ●Cutaneous hyperalgesia : Increase pain sensiblity in skin area surrounding 1ry site of injury(red area) -Mechanism of 1ry hyperalgesia --->Lowering threshold of pain receptors due to local axon reflex -Mechanism of 2ry hyperalgesia --->Convergence facilitation theory not lowering threshold of pain receptors -●Feeling of sensation--->Thalamus -●Localization of senation--->Cerebral cortex ●Visceral pain: -Sharp cut not produce pain sensation but diffuse stimulation of pain receptor produce pain sensation -True Visceral pain--->transmited by autonomic visceral afferent -Parietal Visceral pain--->tranmited by somatic afferents ●Referred pain: Pain felt away its original site -Mechanism--->Convergence projection theory -Cutaneous pain--->Not referred -Deep pain--->May be referred -Visceral pain--->Usually referred ■Covergence facilitation theory --->2ry hyperalgesia ■Convergense projection theory --->Referred pain ●Headache *Very Important* ■Headache is referred pain(الدكتور مأكد عليها) ■Intracranial pain sensitive structures(فالكتاب) ■Causes of intracranial and extracranial headache(فالكتاب) ■Constipation headache cause Intracranial headache(الدكتور مأكد عليها) ●Thalamic syndrome *Very Important* ■Thalamic hyperpathia(دكتور سند مأكد عليها) ●Sensory cortex ■Area 3,1,2--->Perception of sensation ■Area 40--->Poor perception compared to area 3,1,2 ■Area5,7--->Interpretation (Understanding meaning) of sensation ●Syringomyelia ■Loss of sensation of pain , Temperature and crude touch has Jacket distribution as it occurs in lower cervical and upper thoracic regions(دكتور سند مأكد عليها) ■It's example of Dissociated sensory loss as pain , Temperature and crude touch sensations are lost but fine touch and proprioceptive sensation are remain intact(دكتور سند مأكد عليها) ●Tapes dorsalis ■فى أول المرض بيبقى فيه severe pain و loss of fine touch , kinesthetic sensation and vibration sense ليه بقا؟

الإجابة عشان الpain دا بيتنقل عن طريق الunmyelinated C fibers و النوع دا بيتحمل الpressure انما الfine touch , kinesthetic sensation and vibration sense دول بيتنقلو عن طريق الA-beta fibers و ديه مش بتتحمل الpressure و طبعا فآخر المرض هيحصل loss of pain sensation برضو بس بيتأخر شوي عشان تحمل الunmyelinated C fibers ديه ■There is loss of Kinesthetic sensations leads to sensory ataxia(دكتور سند مأكد عليها) ■Mechanisms that maintain equilibruim are Cerebellum , Eye , Kinesthetic sensations و بتحتاج إتنين منهم عالأقل مش هتعرف تتوازن بواحدة بس منهم عشان كدا مثلا لو واحد عنده loss of kinesthetic sensations هو كدا فاقد واحدة منهم لو قولتله يقفل عينيه كدا هيبقى كأنه فقد التانية فا هيقع علطول و دا إسمه Positive Romberg's sign و دا اللى بيأكدلك إن المريض عنده Sensory ataxia ■المريض بيبقى عنده حاجة برضو اسمها Stamping gait لأنه عشان هو مش حاسس بحركة رجله فا بيرفع رجله جامد و بيضرب بيها جامد فالأرض عشان يحس مكان رجله فين بالظبط ●Peripheral neuritis ■Hypothesia and anathesia in distal parts of limps--->Glove and stock(دكتور سند مأكد على ديه) يبقى كدا ■Jacket destribution--->Syringomyelia ■Glove and Stock--->Peripheral neuritis فى النهاية.. متنساش تصلى عالنبى و تدعى لإخواننا فى فلسطين ربنا ينصرهم على أعداؤهم و يصبر قلوب أهالى الشهداء و ربنا يتقبل شهداءهم برحمته فى جنات الفردوس بإذن الله اللهم آمين❤🇵🇸

Ahmed Wagih: ...Notes in Sensory CNS... -Eye responds to : ■Light--->Vision[Adequate] ■Strong box hit or strong blow---> flashes and stars--->Vision[Greater than adequate] -The terminal nerve endings in pacinian corpuscles is unmyelinated and called --->Receptor or Transducer area -The 1st node of Ranvier is called ---> Spike generating region as it's first area where action potential occur -Local anethesia produce effect 1st node of Ranvier not on the receptor so it blocks Action potential but not block receptor potential -Sensory coding : ■Modality discrimination--->Muller's law and labeled line principle ■Site discrimination--->Law of projection ■Intensity discrimination--->Weber-Fechner law -Protopathic sensations are perceived by Thalamus while Epicritic sensations are perceived by Cerebral cortex *But final termination of both is Sensory cortex* ●Pain sensation: -Pain is non specific sensation as it can be caused by chemical,mechanical,thermal stimuli -Pain has specific receptors(Free nerve endings) -Pain receptors don't obey muller's law as most of them are polymodal -Pain receptors are : ■Abundant in skin and some internal organs ■Few in deep tissues and all viscera *Very Important*■Brain itself and Parenchymal tissues like: liver , lung , kidney and spleen have no pain receptors--->Pain insensitive structures(الدكتور مأكد عليها) -Sensitivity of receptors is inversely proportional to its threshold -Chemical transmitter on Fast pain--->Glutamate -Chemical transmitter on Slow pain--->Substance P -A delta fibers blocked by--->Pressure and Hypoxia -Unmyelinated C fibers blocked by--->Local anethesia -Difference in pain senation between different people or individuals not according to pain threshold but according to--->Body reactions to pain -Somatic reflex in body reactions to different types of pain is prepotent reflex--->prevails over other reflexes ●Cutaneous hyperalgesia : Increase pain sensiblity in skin area surrounding 1ry site of injury(red area) -Mechanism of 1ry hyperalgesia --->Lowering threshold of pain receptors due to local axon reflex -Mechanism of 2ry hyperalgesia --->Convergence facilitation theory not lowering threshold of pain receptors -●Feeling of sensation--->Thalamus -●Localization of senation--->Cerebral cortex ●Visceral pain: -Sharp cut not produce pain sensation but diffuse stimulation of pain receptor produce pain sensation -True Visceral pain--->transmited by autonomic visceral afferent -Parietal Visceral pain--->tranmited by somatic afferents ●Referred pain: Pain felt away its original site -Mechanism--->Convergence projection theory -Cutaneous pain--->Not referred -Deep pain--->May be referred -Visceral pain--->Usually referred ■Covergence facilitation theory --->2ry hyperalgesia ■Convergense projection theory --->Referred pain ●Headache *Very Important* ■Headache is referred pain(الدكتور مأكد عليها) ■Intracranial pain sensitive structures(فالكتاب) ■Causes of intracranial and extracranial headache(فالكتاب) ■Constipation headache cause Intracranial headache(الدكتور مأكد عليها) ●Thalamic syndrome *Very Important* ■Thalamic hyperpathia(دكتور سند مأكد عليها) ●Sensory cortex ■Area 3,1,2--->Perception of sensation ■Area 40--->Poor perception compared to area 3,1,2 ■Area5,7--->Interpretation (Understanding meaning) of sensation ●Syringomyelia ■Loss of sensation of pain , Temperature and crude touch has Jacket distribution as it occurs in lower cervical and upper thoracic regions(دكتور سند مأكد عليها) ■It's example of Dissociated sensory loss as pain , Temperature and crude touch sensations are lost but fine touch and proprioceptive sensation are remain intact(دكتور سند مأكد عليها) ●Tapes dorsalis ■فى أول المرض بيبقى فيه severe pain و loss of fine touch , kinesthetic sensation and vibration sense ليه بقا؟

الإجابة عشان الpain دا بيتنقل عن طريق الunmyelinated C fibers و النوع دا بيتحمل الpressure انما الfine touch , kinesthetic sensation and vibration sense دول بيتنقلو عن طريق الA-beta fibers و ديه مش بتتحمل الpressure و طبعا فآخر المرض هيحصل loss of pain sensation برضو بس بيتأخر شوي عشان تحمل الunmyelinated C fibers ديه ■There is loss of Kinesthetic sensations leads to sensory ataxia(دكتور سند مأكد عليها) ■Mechanisms that maintain equilibruim are Cerebellum , Eye , Kinesthetic sensations و بتحتاج إتنين منهم عالأقل مش هتعرف تتوازن بواحدة بس منهم عشان كدا مثلا لو واحد عنده loss of kinesthetic sensations هو كدا فاقد واحدة منهم لو قولتله يقفل عينيه كدا هيبقى كأنه فقد التانية فا هيقع علطول و دا إسمه Positive Romberg's sign و دا اللى بيأكدلك إن المريض عنده Sensory ataxia ■المريض بيبقى عنده حاجة برضو اسمها Stamping gait لأنه عشان هو مش حاسس بحركة رجله فا بيرفع رجله جامد و بيضرب بيها جامد فالأرض عشان يحس مكان رجله فين بالظبط ●Peripheral neuritis ■Hypothesia and anathesia in distal parts of limps--->Glove and stock(دكتور سند مأكد على ديه) يبقى كدا ■Jacket destribution--->Syringomyelia ■Glove and Stock--->Peripheral neuritis فى النهاية.. متنساش تصلى عالنبى و تدعى لإخواننا فى فلسطين ربنا ينصرهم على أعداؤهم و يصبر قلوب أهالى الشهداء و ربنا يتقبل شهداءهم برحمته فى جنات الفردوس بإذن الله اللهم آمين❤🇵🇸

رسومات الهستو يدكاترة

الرسومات العظمة دي من قناة زميلنا في طب المنصورة: محمد عوض هتنزل علي قناته رسومات زي دي لجميع anatomy الموديولات مجانًا وهي مفيدة جدًا وهتسهل عليكم كتير في المذاكرة وربنا يجازيه خير وينفع بالقناة❤ https://t.me/+_CQzZRVCGto3MDNk

معلش أملوا الفورم دا لواحد زميلنا

Hi everyone, this is a study about night eating syndrome among medical students, please feel free to fill the form and help us to reach wide range of medical students The code: 11 Don’t forget to write the code so it can be count, and help me reach the largest nulber we can get together. https://docs.google.com/forms/d/e/1FAIpQLScU0l-ptNkmetxX9ko52WSP3hRnUiwK5WX-U9tVibguJxk2CQ/viewform?usp=pp_url