Healing Ivermectin 📢LIVE CHATS🐴 (DIRT ROAD protocol Horse Paste)
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A place we can gather & help eachother heal, & talk about our issues 24/7 CENSORSHIP FREE join us on our journey to heal w/others. WE'RE NOT DOCTORS (this isnt medical advice) We sell NOTHING.
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پست‌های کانال
| 2 | Investigation of pharmacokinetic interaction between ivermectin and praziquantel after oral administration in healthy dogsZeynep OzdemirHatice Eser FakiKamil UneyBunyamin TrasAuthors and AffiliationsJournal of Veterinary Pharmacology and Therapeutics 42(5):p 497-504, September 2019. | DOI: 10.1111/jvp.12769
The purpose of this study was to determine the pharmacokinetic interaction between ivermectin (0.4 mg/kg) and praziquantel (10 mg/kg) administered either alone or co-administered to dogs after oral treatment.
Twelve healthy cross-bred dogs (weighing 18–21 kg, aged 1–3 years) were allocated randomly into two groups of six dogs (four females, two males) each. In first group, the tablet forms of praziquantel and ivermectin were administered using a crossover design with a 15-day washout period, respectively. Second group received tablet form of ivermectin plus praziquantel. The plasma concentrations of ivermectin and praziquantel were determined by high-performance liquid chromatography using a fluorescence and ultraviolet detector, respectively. The pharmacokinetic parameters of ivermectin following oral alone-administration were as follows: elimination half-life (t1/2λz) 110 ± 11.06 hr, area under the plasma concentration–time curve (AUC0–∞) 7,805 ± 1,768 hr.ng/ml, maximum concentration (Cmax) 137 ± 48.09 ng/ml, and time to reach Cmax (Tmax) 14.0 ± 4.90 hr. The pharmacokinetic parameters of praziquantel following oral alone-administration were as follows: t1/2λz 7.39 ± 3.86 hr, AUC0–∞ 4,301 ± 1,253 hr.ng/ml, Cmax 897 ± 245 ng/ml, and Tmax 5.33 ± 0.82 hr. The pharmacokinetics of ivermectin and praziquantel were not changed, except Tmax of praziquantel in the combined group. In conclusion, the combined formulation of ivermectin and praziquantel can be preferred in the treatment and prevention of diseases caused by susceptible parasites in dogs because no pharmacokinetic interaction was determined between them.
Basically
study examining the co-administration of ivermectin and praziquantel in healthy dogs found no significant pharmacokinetic interactions between the two medications, indicating that the drugs do not interfere with each other's processing in the body. The findings suggest that a combined formulation of these parasite treatments is clinically viable and safe for routine veterinary use.
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| 3 | Ivermectin Augments the In Vitro and In Vivo Efficacy of Cisplatin in Epithelial Ovarian Cancer by Suppressing Akt/mTOR Signaling
Xiaohong Zhang
Tingting Qin
Zhengyan Zhu
Fan Hong
Yang Xu
Xiongjie Zhang
Xiaohong Xu
Aiping Ma
Authors and Affiliations
American Journal of the Medical Sciences 359(2):p 123-129, February 2020. | DOI: 10.1016/j.amjms.2019.11.001
Abstract
ABSTRACT
Background:
The poor outcomes in epithelial ovarian cancer necessitate new treatments. In this work, we systematically analyzed the inhibitory effects of ivermectin and the molecular mechanism of its action in ovarian cancer.
Methods:
The effects of ivermectin alone and its combination with cisplatin on growth and survival were examined using cultured ovarian cancer cells and a xenograft mouse model. The molecular mechanism of action of ivermectin, focusing on Akt/mTOR signaling, was elucidated.
Results:
Ivermectin arrested growth in the G2/M phase and induced caspase-dependent apoptosis in ovarian cancer, regardless of specific cellular and molecular differences. Ivermectin significantly augmented the inhibitory effect of cisplatin on ovarian cancer cells in a dose-dependent manner. Mechanistically, ivermectin suppressed the phosphorylation of key molecules in the Akt/mTOR signaling pathway in ovarian cancer cells. In addition, overexpression of constitutively active Akt restored ivermectin-induced inhibition of Akt/mTOR, growth arrest and apoptosis. In an ovarian cancer xenograft mouse model, ivermectin alone significantly inhibited tumor growth. In combination with cisplatin, tumor growth was completely reversed over the entire duration of drug treatment without any toxicity. Furthermore, the concentrations of ivermectin used in our study are pharmacologically achievable.
Conclusions:
Our work suggests that ivermectin may be a useful addition to the treatment armamentarium for ovarian cancer and that targeting Akt/mTOR signaling is a therapeutic strategy to increase chemosensitivity in ovarian cancer.
Copyright © 2020 by the Southern Society for Clinical Investigation.
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| 4 | Repurposing Ivermectin to augment chemotherapy's efficacy in osteosarcoma
B Hu
H Tan
L Yu
Q Liao
W Guo
Authors and Affiliations
Human & Experimental Toxicology 41:p 09603271221143693, 2022. | DOI: 10.1177/09603271221143693
Abstract
Abstract
Background:
Osteosarcoma is the most frequent malignant bone malignancy and the current treatments are ineffective. Ivermectin, an anti-protozoal drug, has been shown to have anti-cancer activity. This work investigated the potential of repurposing ivermectin to augment chemotherapy's efficacy in osteosarcoma.
Methods:
Proliferation, migration and apoptosis assays were performed in ivermectin-treated osteosarcoma cells. Combination studies were performed. Osteosarcoma xenograft mouse model was established to investigate the in vivo efficacy of ivermectin. Intracellular reactive oxygen species (ROS) and mitochondrial superoxide, membrane potential, ATP, 8-OHdG level, protein carbonylation and lipid peroxidation were determined after ivermectin treatment.
Results:
Ivermectin was effective and acted synergistically with doxorubicin in osteosarcoma cells regardless of cellular origin and genetic profiling. This was achieved through suppressing inhibiting growth and migration, and inducing caspase-dependent apoptosis. Ivermectin also significantly inhibited osteosarcoma growth in vivo and its combination with doxorubicin resulted in much greater efficacy than doxorubicin alone. Importantly, the effective dose of ivermectin was clinically feasible and did not cause significant toxicity in mice. Mechanistical analysis showed that ivermectin induced oxidative stress and damage, and mitochondrial dysfunction.
Conclusions:
Our findings indicate that ivermectin has utility in treating patients with osteosarcoma, especially those resistant to chemotherapy.
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| 5 | Clinical efficacy and safety of topical versus oral ivermectin in treatment of uncomplicated scabies
Hesham M. Ahmad
Eman S. Abdel-Azim
Rasha T. Abdel-Aziz
Authors and Affiliations
Dermatologic Therapy 29(1):p 58-63, January-February 2016. | DOI: 10.1111/dth.12310
Abstract
ABSTRACT:
Many medications are available for scabies treatment including oral and topical ivermectin. However, studies comparing these two forms as a scabies treatment are few. This study compares efficacy and safety of topical versus oral ivermectin as scabies treatment. The study included 62 confirmed uncomplicated scabies patients, divided into: Group I (32 patients, received topical ivermectin) and Group II (30 patients, received oral ivermectin). Patients were assessed, clinically and by KOH smear at 1, 2 and 4 weeks. Treatment was repeated after one week in patients with persistent infection. Adverse events were recorded. Most patients (87.5% and 73.5% in group I and group II respectively) were symptom free after a single treatment. A second treatment was required in 4 patients of group I and 8 patients of group II. However, 2 weeks after treatment symptoms and signs completely resolved in all cases with no recurrence at 4 weeks. This study suggests that both topical and oral ivermectin are safe and equally effective in treatment of uncomplicated scabies. Single treatment, whether topical or oral, is associated with high cure rate in a week post treatment. However, repeating treatment after one week may be required to achieve 100% cure.
Copyright © 2016 Blackwell Science, Inc.
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| 6 | https://youtube.com/shorts/luzdimuvZ7w?is=kD2PlREURnSPie-N | 239 |
| 7 | Ingredient-List-for-Vaccines.pdf
https://ks.childrenshealthdefense.org/wp-content/uploads/Ingredient-List-for-Vaccines.pdf | 220 |
| 8 | X Post
💉SOUTH CAROLINA: THEY VACCINATED 1,200 KIDS. THEN THE OUTBREAK HAPPENED.
South Carolina just dropped a bill to remove the religious exemption for the MMR vaccine, eliminating exemptions for kids in childcare, school, or college.
Why? Because there's a measles outbreak in Spartanburg County. About 95% of the cases are happening there.
But here's what they're not telling you:
That same county just did a massive vaccination push. They vaccinated over 1,200 kids aged 6 months to 11 months.
The MMR vaccine is a live virus. About 7% of kids who get it will develop a rash and fever, which meets the clinical definition of measles.
And those vaccinated kids? They can spread it.
@NVICAdvocacy joined us to talk about this and said:
Here's the question everyone should be demanding an answer to: Are we seeing a wild-type measles outbreak, or are we seeing vaccine-strain measles spreading from the 1,200 kids they vaccinated?
They're not testing. They're not distinguishing between wild-type and vaccine-strain. They're not advertising whether they're even checking.
This is a highly imperfect vaccine. It doesn't stop subclinical spread. It will not - and CANNOT - ever produce herd immunity. That's just the reality.
But instead of acknowledging that, they're deflecting and blaming the unvaccinated. And now they want to eliminate religious exemptions and mandate the MMR for everyone.
1,200 kids vaccinated. Then an outbreak in the same county. And the solution is to vaccinate MORE kids and remove exemptions?
The story is in the nuance. The vaccine is live. The vaccine can cause measles symptoms. The vaccinated can spread it. And they're not testing to distinguish.
But they'll try to take away your right to refuse it anyway.
This is how policy gets made. Crisis. Reaction. Mandate.
Don't let it slide this time... | 224 |
| 9 | The drugs, which come in tablets and lozenges, are routinely prescribed to children in the U.S. as a means to prevent cavities. They can be prescribed to babies as young as 6 months old. 😳 https://childrenshealthdefense.org/defender/fda-fluoride-prescription-drugs-for-kids-off-market/ | 128 |
| 10 |  بدون متن... | 186 |
| 11 | A groundbreaking study reveals a potential link between beekeeping, longevity, and the consumption of bee-related products like honey, according to information published by the National Library of Medicine. Researchers found significant associations between beekeepers and longer telomeres, indicative of increased lifespan compared to non-beekeepers.
Telomere length, a marker for biological aging, was notably longer in beekeepers, especially among those who consumed bee products regularly. The study suggests that frequent intake of bee products, including honey, propolis, and royal jelly, may contribute to maintaining telomere length, thus potentially delaying aging and associated diseases. https://pmc.ncbi.nlm.nih.gov/articles/PMC4450150/ | 108 |
| 12 | Did you know beekeepers live longer? I would love to try bee bed therapy one day! 🐝 https://youtube.com/shorts/js1q9NTSgIY?si=SPMD9bl0ICJ8sMVI | 104 |
| 13 |  Know the food you eat....
Join:✅️💚
https://t.me/HEALINGCHATS | 322 |
| 14 |  Know The Food You Eat...
Everything Is Being Contaminated Since the Food, The Water Even the Air We Breathe, The Whole Earth Is Being Tainted
Join us 👉 Https://t.me/HumanResistanceAllianceHRA | 1 |
| 15 |  بدون متن... | 122 |
| 16 | Oldie but goody....NEVER FORGET! | 208 |
| 17 |  بدون متن... | 130 |
| 18 |  When your immunity drops the microscopic critters come out to play. Dermatologists are 100% in on the grift too!!! Rosacea = mites/parasites. Acne is also another common skin problem that is often caused by Bartonella (Cat scratch/Lyme Disease bacteria) and never properly treated, but they make billions band-aiding them with useless products and prescriptions! | 197 |
| 19 |  بدون متن... | 172 |
| 20 | Join💚✅️https://t.me/HEALINGCHATS | 170 |
