Emergency notes🫀
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قناة مهتمة بنشر معلومات وملاحظات يحتاجها كل طالب او طبيب طوارئ …
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3 402
Hematuria + proteinuria + hypertension + periorbital edema
👉 Glomerulonephritis
Hematuria+ hemoptysis + Rapid deterioration of renal function
👉 Goodpasture syndrome
Or Granulomatosis with polyangiitis depend on age
Hematuria +Chronic sinusitis or nasal ulcer + Epistaxis
👉 Granulomatosis with polyangiitis
Hematuria + cramping abdominal pain + painful palpable purpura+ arthralgia
👉 Henoch Schonlein purpura
Hematuria+ resistant asthma + eosinophilia(>1,000)+neuropathy
👉 Churg Strauss
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🤡Beta blockers, calcium channel blockers, and nitrates can cause false negative results on exercise and pharmacologic stress testing and should be held for 48 hours prior to testing.
However, aspirin, statins, and lisinopril are not known to affect stress testing results.
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A 64-year-old man is admitted to the hospital due to intermittent palpitations and lightheadedness for the past several days. The patient has had no chest pain or shortness of breath. He has a history of coronary artery disease with prior myocardial infarction and percutaneous coronary intervention. Medications include metoprolol, lisinopril, aspirin, clopidogrel, and rosuvastatin. Echocardiogram reveals mild left ventricular dilation, a left ventricular ejection fraction of 30%, and no major valvular abnormality. Serum potassium is 4.2 mEq/L, and magnesium is 1.9 mg/dL. On day 2 of hospitalization, the patient develops sudden-onset and sustained palpitations. Blood pressure is 122/60 mm Hg and pulse is 120/min. He is alert and does not appear to be in acute distress. The lungs are clear bilaterally. ECG rhythm strip is shown in the exhibit. Which of the following is the best next step in management of this patient
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Treatment of sustained monomorphic VT depends on the patient's clinical status.
Patients with pulseless VT are treated with immediate defibrillation.
Patients with a pulse but evidence of hemodynamic instability (eg, hypotension, angina, confusion) are treated with immediate synchronized electrical cardioversion ().
In hemodynamically stable patients such as this one, pharmacologic cardioversion to sinus rhythm should be attempted. Intravenous amiodarone is usually the preferred agent; other agents (eg, procainamide, sotalol, lidocaine) are also sometimes used
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Tetrology of Fallot
"Don't DROP the baby":
Defect (VSD)
Right ventricular hypertrophy
Overriding aorta
Pulmonary stenosis
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Respiratory depression inducing drugs
"STOP breathing":⭐️
Sedatives and hypnotics
Trimethoprim
Opiates
Polymyxins
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Lithium: side effects
LITHIUM:⭐️
Leukocytes Increased (leukocytosis)
Tremors
Hypothyroidism
Increased Urine
Moms beware (teratogenic)
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Antiarrhythmics: class III members
BIAS:⭐️
Bretylium
Ibutilide
Amiodarone
Sotalol
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Beta blockers: members
"The NEPAL Prime Minister":💡
Timolol
Nadolol
Esmolol
Pindolol
Atenolol
Labetalol
Propranolol
Metoprolol
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The mnemonic “ELEVATION”, can help you remember STEMI mimics
- Electrolytes (Hyperkalemia)
- Left Bundle Branch Block
- Early Repolarization
- Ventricular Hypertrophy (Left)
- Aneurysm (Ventricular)
- Thailand (Brugada Syndrome)
- Inflammation (Pericarditis)
- Osborne (J) Waves (hypothermia)
- Non-Ischemic Vasospasm
