Dr. Ben Tapper

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The also removed my post when I asked Elon about it…

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Nanomaterials can be transported by monocyte-lineage cells to DLNs, blood and spleen, and, similarly to HIV, may use CCL2-dependent mechanisms to penetrate the brain. This occurs at a very low rate in normal conditions explaining good overall tolerance of alum despite its strong neurotoxic potential. However, continuously escalating doses of this poorly biodegradable adjuvant in the population may become insidiously unsafe, especially in the case of overimmunization or immature/altered blood brain barrier or high constitutive CCL-2 production. http://www.biomedcentral.com/1741-7015/11/99 Advertisement The Role of Spiritual Enlightenment a... An Analogy with Butterfly Metamorphosis Today’s discourse touches upon the in... Share this: TwitterFacebook Loading... Related my response to a friend who fears vaccination and is unsure what to do. May 5, 2013 In "thinking free" Pediatricians Receive Regular Requests for Alternative Child Immunization Schedules..really!!? January 20, 2013 In "thinking free" what excactly is in that? March 8, 2013 In "thinking free" • Posted by the referbished rogue in vaccination Post navigation ← regarding tdap and pregnancy..is an autism coverup to blame? Read the back of your toothpaste – fluoride IS poison. if there’s doubt – GET IT OUT! → 57 thoughts on “my list of peer reviewed vaccine research” sandy fleming May 4, 2013 at 12:59 am this is amazing to say the least. Reply the referbished rogue May 4, 2013 at 1:34 am I am glad that you think so 🙂 I hope that it comes in handy for you in the future Reply Jyothi May 4, 2013 at 1:40 pm Thanks so much for putting this together! Have bookmarked and will share! Reply the referbished rogue May 4, 2013 at 1:44 pm yourwelcome! thank you so much..one person at a time.. getting the word out is how it started..and I am so thankful to whoever it was that spoke out first 🙂 I feel honored to have been “awakened”.. God bless. – briana Reply Ravi May 4, 2013 at 4:38 pm Great information all in one place. Thanks for posting and definitely sharing. BTW, the reason Polio disappeared from the map was also because the CDC renamed the disease to Aseptic Viral meningitis and also Acute Flaccid Paralysis. You can see that just about these two diseases were brought forth the numbers for these went up while the numbers for polio disappeared. Not sure if this was mentioned in the article (did not read all of it yet :)). Thanks again Reply the referbished rogue May 4, 2013 at 5:07 pm Ravi, thank you so very much for reminding me about this! unfortunately, this list is what survived a computer crash and then a new computer. I lost so so so much stuff..so much knowledge forgotten..but, I don’t mind it all that much..i look forward to finding them all again. The renaming of polio scam did not make it on my list, I don’t think. I remember reading about it a long time ago on an ‘InsideVaccines’ post and it led me to find the studies and such..but apparently that lead is just one of many lost 😦 but now that you have reminded me of it..im off to do some research and will hopefully be adding this to my list tonight. any thing else you’d like to remind me of? ha 🙂 God bless – briana Reply Sheri Nakken, former RN, MA, Hahnemannian Homeopath November 17, 2013 at 7:01 pm and also wherever polio was, there was DDT use http://www.wellwithin1.com/Polio.West.htm Gayl H May 4, 2013 at 6:32 pm What a great start! Like you, I have read 100s if not thousands of articles, and have been trying to put together a compendium such as this on paper/computer, but didn’t think of archiving them like this! Excellent “abstracting” of the articles as well! This is a tremendous service to all who are trying to research vaccine safety 🙂 Reply the referbished rogue May 4, 2013 at 7:40 pm I am just overwhelmed by all the kind comments.. thank you Gayl H. When ever you get yours completed I would love to take a peek. if you remember me then..let me know 🙂 -briana Reply

The predominance in these outbreaks of infected people of university age not only highlights an environment with potential for increased transmission but also raises questions about the efficacy of the MMR vaccine. The people affected may represent a “lost cohort” who do not have immunity from natural mumps infection and were not offered a 2-dose schedule. Given the current level of mumps activity around the world, clinicians should remain vigilant for symptoms of mumps. http://www.ncbi.nlm.nih.gov/pubmed/16940266   Persistence of maternal antibody in infants beyond 12 months: Mechanism of measles vaccine failure A serologic study was made in 34 children immunized against measles at the age of 12 months. Using a sensitive virus neutralization test, it was found that many of the children had pre-existing maternal antibody to measles virus. (this was written in 1977 back when mothers were actually passing immunity to their children..this is just an example of natural immunity being passed from mother to child..something that vaccination cannot and will not ever do.) http://www.sciencedirect.com/science/article/pii/S0022347677810214 The study, which analyzed data from 2009-2011, found that white, college-educated mothers over the age of 35 were most likely to report that they had delayed or skipped immunizations for their children. There’s no consensus as to why that is the case, Young said. [hmmm..lets try to help them come to a clear consensus.. could intellence level be a factor? could age play a role because mothers over 35 have had more time to witness what vaccination can do?] Read more here: http://www.adn.com/2013/04/22/2875131/more-alaskans-hesitant-about-vaccines.html#storylink=cpy “A Positive Association found between Autism Prevalence and Childhood Vaccination uptake across the U.S. Population” The reason for the rapid rise of autism in the United States that began in the 1990s is a mystery. Although individuals probably have a genetic predisposition to develop autism, researchers suspect that one or more environmental triggers are also needed. One of those triggers might be the battery of vaccinations that young children receive. Using regression analysis and controlling for family income and ethnicity, the relationship between the proportion of children who received the recommended vaccines by age 2 years and the prevalence of autism (AUT) or speech or language impairment (SLI) in each U.S. state from 2001 and 2007 was determined. A positive and statistically significant relationship was found: The higher the proportion of children receiving recommended vaccinations, the higher was the prevalence of AUT or SLI. A 1% increase in vaccination was associated with an additional 680 children having AUT or SLI. Neither parental behavior nor access to care affected the results, since vaccination proportions were not significantly related (statistically) to any other disability or to the number of pediatricians in a U.S. state. The results suggest that although mercury has been removed from many vaccines, other culprits may link vaccines to autism. Further study into the relationship between vaccines and autism is warranted   full text: http://www.theoneclickgroup.co.uk/documents/vaccines/Vaccine%20and%20Autism%20correlation%20US%202011%20J%20Tox%20Env%20Health.pdf     “CDC officials discuss neurological damage from vaccines in secret meeting – Simpsonwood” You can read this clearly for yourself if you access the pdf transcript that was obtained via FOIA http://therefusers.com/refusers-newsroom/cdc-officials-discuss-neurological-damage-from-vaccines-in-secret-meeting-simpsonwood/#.UYM4l07D_IV “The odds of having a history of asthma was twice as great among vaccinated subjects than among unvaccinated subjects  The odds of having had any allergy-related respiratory symptom in the past 12 months was 63% greater among vaccinated subjects than unvaccinated subjects” http://www.ncbi.nlm.nih.gov/pubmed/10714532

There were 415 arthritis, 166 rheumatoid arthritis, 130 myelitis, 4 SLE, 100 optic neuritis, 101 GBS, 29 glomerulonephritis, 283 pancytopenia/thrombocytopenia, and 183 MS events reportedfollowing HBV A total of 465 positive re-challenge adverse events were observed following adult HBV that occurred sooner and with more severity than initial adverse event reports. A case-report of arthritis occurring in identical twins was also identified. [personal note: between 1 and 10 percent of adverse events are actually reported according to the FDAs David Kessler) http://www.ncbi.nlm.nih.gov/pubmed/15638050 Viral proteins having molecular mimicry with self-proteins in the CNS can prime genetically susceptible individuals. Once this priming has occurred, an immunologic challenge could result in disease through bystander activation by cytokines. Read More: http://informahealthcare.com/doi/abs/10.1080/08916930500484799 Nevertheless, allergy and, to a lesser extent, autoimmunity have repeatedly been described or suspected as rare adverse consequences of human vaccines. The mechanisms of these adverse reactions are ill-elucidated, if at all. No animal models have been adequately standardized and validated to predict the risk of allergy and autoimmunity associated with vaccines. However, a number of existing models can be considered for use, but need refinement to be applied to vaccine evaluation. Finally, because the preclinical safety evaluation has not received much attention in the past, efforts should be paid to design specific and cost-effective procedures to meet the current expectations. http://www.sciencedirect.com/science/article/pii/S0300483X02000562 After reviewing the 27 cases of vasculitis after hepatitis B vaccination reported in the current literature, the authors suggest that, in some cases, vaccination may be the triggering factor for vasculitis in individuals with a genetic predisposition. Physicians should be aware of this possible association. http://www.sciencedirect.com/science/article/pii/S0953620508000770 Mumps resurgences in the United States: A historical perspective on unexpected elements. . The 2006 epidemic followed this pattern, with two unique variations: it was preceded by a period of very high vaccination rates and very low disease incidence and was characterized by two-dose failure rates among adults vaccinated in childhood. Data from the past 80 years suggest that preventing future mumps epidemics will depend on innovative measures to detect and eliminate build-up of susceptibles among highly vaccinated populations http://www.ncbi.nlm.nih.gov/pubmed/19815120 Subacute thyroiditis and dyserythropoesis after influenza vaccination suggesting immune dysregulation. http://www.ncbi.nlm.nih.gov/pubmed/22111471 However, a 2006 epidemic involved >5700 cases nationwide, with many reported among fully vaccinated college students.. A large mumps outbreak occurred despite high two-dose vaccination coverage in a population most of whom had received the second dose >10 years before. Two-dose vaccine effectiveness was similar to previous one-dose estimates. Further studies are needed to examine the persistence of two-dose mumps vaccine-induced immunity and to determine whether US mumps elimination can be achieved with the current vaccination strategy. http://www.ncbi.nlm.nih.gov/pubmed/18539365 The first outbreak involved 13 high-school students (median age 14 yr): 9 who had previously received 2 doses of measles-mumps-rubella vaccine (MMR) and 4 who received a single dose. The second outbreak comprised 19 cases of mumps among students and some staff at a local university (median age 23 yr), of whom 18 had received only 1 dose of MMR (the other received a second dose). The viruses identified in the outbreaks were phylogenetically similar and belonged to a genotype commonly reported in the UK. The virus from the second outbreak is identical to the strain currently circulating in the UK and United States. INTERPRETATION:

Conclusions: Our results suggest that vaccines other than influenza vaccine can be associated with GBS. Vaccination-related GBS results in death or disability in one fifth of affected individuals, which is comparable to the reported rates in the general GBS population http://journals.lww.com/jcnmd/Abstract/2009/09000/Guillain_Barre_Syndrome_after_Vaccination_in.1.aspx Autoimmune reactions to vaccinations may rarely be induced in predisposed individuals by molecular mimicry or bystander activation mechanisms. Autoimmune reactions reliably considered vaccine-associated, include Guillain-Barré syndrome after 1976 swine influenza vaccine, immune thrombocytopenic purpura after measles/mumps/rubella vaccine, and myopericarditis after smallpox vaccination, whereas the suspected association between hepatitis B vaccine and multiple sclerosis has not been further confirmed, even though it has been recently reconsidered, and the one between childhood immunization and type 1 diabetes seems by now to be definitively gone down. Larger epidemiological studies are needed to obtain more reliable data in most suggested associations. Read More: http://informahealthcare.com/doi/abs/10.3109/08830181003746304 Vaccines, in several reports were found to be temporally followed by a new onset of autoimmune diseases. The same mechanisms that act in infectious invasion of the host, apply equally to the host response to vaccination. It has been accepted for diphtheria and tetanus toxoid, polio and measles vaccines and GBS. Also this theory has been accepted for MMR vaccination and development of autoimmune thrombocytopenia, MS has been associated with HBV vaccination. Read More: http://informahealthcare.com/doi/abs/10.1080/08916930500050277 Hepatitis B infection is one of the most important causes of acute and chronic liver disease. During the 1980s, genetically engineered hepatitis B vaccines (HBVs) were introduced in the United States. A large-series of serious autoimmune conditions have been reported following HBVs, despite the fact that HBVs have been reported to be “generally well-tolerated.” A case-control epidemiological study was conducted to evaluate serious autoimmune adverse events prospectively reported to the vaccine adverse events reporting system (VAERS) database following HBVs, in comparison to an age, sex, and vaccine year matched unexposed tetanus-containing vaccine (TCV) group for conditions that have been previously identified on an a priori basis from case-reports. Adults receiving HBV had significantly increased odds ratios (OR) for multiple sclerosis (OR = 5.2, p < 0.0003, 95% Confidence Interval (CI) = 1.9 – 20), optic neuritis (OR = 14, p < 0.0002, 95% CI = 2.3 – 560), vasculitis (OR = 2.6, p < 0.04, 95% CI = 1.03 – 8.7), arthritis (OR = 2.01, p < 0.0003, 95% CI = 1.3 – 3.1), alopecia (OR = 7.2, p < 0.0001, 95% CI = 3.2 – 20), lupus erythematosus (OR = 9.1, p < 0.0001, 95% CI = 2.3 – 76), rheumatoid arthritis (OR = 18, p < 0.0001, 95% CI = 3.1 – 740), and thrombocytopenia (OR = 2.3, p < 0.04, 95% CI = 1.02 – 6.2) in comparison to the TCV group. Minimal confounding or systematic error was observed. Despite the negative findings of the present study regarding the rare serious adverse effects of HBVs, it is clear that HBV does, indeed, offer significant benefits, but it is also clear that chances of exposure to hepatitis B virus in adults is largely life-style dependent. Adults should make an informed consent decision, weighing the risks and benefits of HBV, as to whether or not to be immunized http://www.ncbi.nlm.nih.gov/pubmed/16206512 HBV was associated with a number of serious conditions and positive re-challenge or significant exacerbation of symptoms following immunization.

found to be homologous to myelin basic protein (23). 2) Another coincidental infection: Most of the vaccine recipients are at high risk for infection with EBV, C M V and HTLV 3, that also can cause demyelinating disease (18). 3) Immune complex disease: Five cases of GBS have been reported in patients suffering from infection with HBV. In the acute phase of GBS, immune complexes containing hepatitis B surface antigen were found in the serum and cerebrospinal fluid, but not in the sural nerve. Those immune complexes were not present when the hepatitis was first detected, but only after the appearance of neurological symptoms, and disappeared when the inflammatory phase of the disease had ended (24, 25). Immune complexes without a known antigen were found in other cases of GBS in various organs. The immune complexes can transfer through the blood-nerve barrier and may be deposited in the endonerium and injure nerve fibers (25). Treatment with plasmapheresis or IVIG may eliminate those immune complexes. R e c e n t l y, the presence of glycolipid (ganglioside) specific antibodies has been found to be associated with neurological disease, in particular with GBS. The pathogenic potential of these antibodies has remained unclear. Several mechanisms by which anti-ganglioside antibodies may exert their potential pathogenic effect have been proposed. Direct binding of anti-ganglioside antibodies to axon or Schwann cells might disturb ion fluxes and cause partial nerve conduction block (26). Naturally occurring antibodies crossre – acting with gangliosides may become pathogenic after affinity maturation and class switching initiated by preceding infection. The hepatitis B vaccine has been used routinely for almost 20 years. Most of the side effects are local or transient minor reactions. The rate of the adverse events is 1 in 15,500 doses. Major reactions are rare and include variable autoimmune phenomena: erythema nodosum, lichen planus, acute urticaria, polyarthritis, including rheumatoid arthritis and reactive arthritis, vasculitis, glomerulonephritis, Evan’s syndrome and thrombocytopenic purpura. Neurological complications include acute cerebellar ataxia and autoimmune demyelinating disorders including multiple sclerosis, transverse myelitis and GBS (27). These reactions are sporadic and there is no clear evidence that the rate of GBS or multiple sclerosis is more common among the vaccinated population. Hepatitis B vaccine is important and, according to the available data, the prevention of hepatitis B outweighs the rare incidence of diseases reported after vaccination. Further animal studies and evaluation of the risk factors for these adverse effects are indicated. http://www.clinexprheumatol.org/article.asp?a=2492 Guillain-Barre Syndrome after Vaccination in United States: Data From the Centers for Disease Control and Prevention/Food and Drug Administration Vaccine Adverse Event Reporting System (1990-2005) Methods: We used data for 1990 to 2005 from the Vaccine Adverse Event Reporting System, which is a cooperative program of the Centers for Disease Control and Prevention and the US Food and Drug Administration. Results: There were 1000 cases (mean age, 47 years) of GBS reported after vaccination in the United States between 1990 and 2005. The onset of GBS was within 6 weeks in 774 cases, >6 weeks in 101, and unknown in 125. Death and disability after the event occurred in 32 (3.2%) and 167 (16.7%) subjects, respectively. The highest number (n = 632) of GBS cases was observed in subjects receiving influenza vaccine followed by hepatitis B vaccine (n = 94). Other vaccines or combinations of vaccines were associated with 274 cases of GBS. The incidence of GBS after influenza vaccination was marginally higher in subjects <65 years compared with those ≥65 years (P = 0.09); for hepatitis vaccine, the incidence was significantly higher (P < 0.0001) in the <65 group. Death was more frequent in subjects ≥65 years compared with those <65 years (P < 0.0001).

cells with cross-reactivity to nervesheath components (4). The disease is related in most cases to respiratory or gastrointestinal infections and vaccines, resulting in demyelination or axonal degeneration (2). The target of the immune attack differs with the clinical subtypes of GBS (3). Rarely is GBS related to Hodgkin’s lymphoma (6) or autoimmune disease such as systemic lupus erythematosus (7). Infection with the following microorganisms can cause GBS: Campylobac – ter jejuni, in 25-41% of GBS patients, Epstein-Barr virus, cytomegalovirus (2), HIV infection, Mycoplasma pneu – moniae, shigella, clostridium (8), and Haemophilus influenzae (9). Vaccines reportedly related to the appearance of GBS include influenza, tetanus toxoid, BCG, rabies, smallpox, mumps, rubella, oral poliovirus vaccine, hepatitis B vaccines, either plasma- derived or recombinant vaccine and diphtheria vaccine (10). The influenza vaccine in 1976 (“swine flue” or New Jersey 76) caused a 4- to 8-fold increase in the rate of GBS occurring 6-8 weeks after vaccination (11,12). Subsequent studies of influenza-vaccinated patients showed no increase in the GBS rate (13). In a review of the English literature another 19 cases of hepatitis B vaccination were reported to precede the symptoms of GBS (14-22) (Table I). T h e plasma-derived hepatitis B vaccine became commercially available in June 1982. Shaw et al. (15) documented the first 3 years of postmarketing surveillance for neurologic adverse events after vaccination among 850,000 persons, mostly health workers, who received the HBV vaccine. Nine cases of GBS were reported up to 7 weeks after vaccination. One case was reported as atypical and 5 cases were compatible with a viral infection before the appearance of the neurological symptoms. GBS was reported as occurring significantly more often then expected when compared with the Center of Disease Control GBS background rate (11), but not when compared with the Olmsted County rate (23). The authors calculated that, taking into account age, sex and under-reporting, the rate of GBS was slightly higher in the vaccinated group, but concluded that no definite epidemiologic association could be made. Mcmahon et al. (17) determined the incidence of adverse reactions from the plasma-derived hepatitis B vaccine in Alaska. Out of 43,618 subjects who received 101,360 injections, 2 patients developed GBS 3 and 9 months after the last injection. Their conclusion was that the vaccine was safe and that the incidence of GBS was not increased. The authors claimed that the adverse events caused by the plasma-derived HBV vaccine are due to the preservative material thimerosal, a mercurial compound that was found to be neurotoxic and is not included in the HBV vaccines since 1999 and to aluminium hydroxide, used as an adjuvant. Both compounds were also used in the recombinant vaccine. In addition to our patient, 8 case reports of GBS after hepatitis B vaccine have been reported (14,16,18-22), 3 of them after receiving the yeast derived recombinant DNA hepatitis B vaccine. One of the patients died after a multiorgan failure, septic shock and adult respiratory distress syndrome. A n e u r opathologic examination revealed an inflammatory cell infiltrate in the gray matter especially in the anterior horn of the spinal cord, and small foci of macrophages in the long tracts. Most of the cells appeared around blood vessels, but were also found in the parenchyma, close to nerve cells (21). The pathogenesis of hepatitis B vaccine associated with GBS is not clear. The following mechanisms are suggested: 1 ) Molecular mimicry: As in other autoimmune disorders appearing after vaccination, molecular mimicry is suspected. Hepatitis B surface protein may provoke an autoimmune attack on a similar protein present in the nerve cells. In molecular mimicry involving T lymphocytes these cells recognize their antigen as peptide- bound to MHC molecule. The microbial antigen has the same shape as a self antigenic epitope bound to the same MHC molecule. The DNA sequence of HBV w a s

We have retrospectively analyzed the medical records of 114 patients, from different centers in the USA, diagnosed with immune-mediated diseases following immunization with hepatitis-B vaccine (HBVv). All patients in this cohort sought legal consultation. Of these, 93/114 patients diagnosed with disease before applying for legal consultation were included in the study. All medical records were evaluated for demographics, medical history, number of vaccine doses, peri-immunization adverse events and clinical manifestations of diseases. In addition, available blood tests, imaging results, treatments and outcomes were recorded. Signs and symptoms of the different immune-mediated diseases were grouped according to the organ or system involved. ASIA criteria were applied to all patients. RESULTS: The mean age of 93 patients was 26.5 ± 15 years; 69.2% were female and 21% were considered autoimmune susceptible. The mean latency period from the last dose of HBVv and onset of symptoms was 43.2 days. Of note, 47% of patients continued with the immunization program despite experiencing adverse events. Manifestations that were commonly reported included neuro-psychiatric (70%), fatigue (42%) mucocutaneous (30%), musculoskeletal (59%) and gastrointestinal (50%) complaints. Elevated titers of autoantibodies were documented in 80% of sera tested. In this cohort 80/93 patients (86%), comprising 57/59 (96%) adults and 23/34 (68%) children, fulfilled the required criteria for ASIA. CONCLUSIONS: Common clinical characteristics were observed among 93 patients diagnosed with immune-mediated conditions post-HBVv, suggesting a common denominator in these diseases. In addition, risk factors such as history of autoimmune diseases and the appearance of adverse event(s) during immunization may serve to predict the risk of post-immunization diseases. The ASIA criteria were found to be very useful among adults with post-vaccination events. The application of the ASIA criteria to pediatric populations requires further study. http://www.ncbi.nlm.nih.gov/pubmed/22235045 vaccination may be associated with autoimmune disease (title of article press link to read) http://www.feingold.org/Research/PDFstudies/Tishler2004-open.pdf Antigen-presenting Cell Activation: a Link Between Infection and Autoimmunity? The onset of autoimmune diseases such as type I diabetes and multiple sclerosis is often thought to be associated with infection. This has led to studies of molecular mimicry between infectious agents and the self-antigens associated with autoimmunity. Despite many claims, however, a single causative infectious agent for autoimmunity has not been found. An alternative possibility is that many infectious agents are capable of non-specifically enhancing the likelihood of an autoimmune attack. Here we show how infectious agents may activate antigen-presenting cells leading to the activation of autoreactive T cells by otherwise innocuous antigens. The mechanism of activation involves upregulation of co-stimulatory molecules on the antigen-presenting cell resulting in a lowering of the threshold required for activation. These results help explain how diverse infectious agents could cause autoimmune disease in susceptible individuals. http://www.sciencedirect.com/science/article/pii/S0896841100904980 Pemphigus is an autoimmune blistering disease caused by autoantibodies against epithelial intercellular components. Its etiology is unknown, and neoplasms, antecedent infections or medications are considered possible triggering factors for the disease in some cases. We describe the first case of pemphigus following a hepatitis B virus vaccination. We suggest that in some cases vaccination may be the triggering factor for pemphigus in genetically predisposed individuals and physicians should be aware of this possible association. Read More: http://informahealthcare.com/doi/abs/10.1080/08916930400027078 Discussion There is increasing evidence that GBS is an autoimmune disease. Various autoantibodies to gangliosides were described in GBS patients (4,5), and T

Because many autistic children harbor elevated levels of measles antibodies, we conducted a serological study of measles-mumps-rubella (MMR) and MBP autoantibodies. Using serum samples of 125 autistic children and 92 control children, antibodies were assayed by ELISA or immunoblotting methods. ELISA analysis showed a significant increase in the level of MMR antibodies in autistic children. Immunoblotting analysis revealed the presence of an unusual MMR antibody in 75 of 125 (60%) autistic sera but not in control sera. This antibody specifically detected a protein of 73-75 kD of MMR. This protein band, as analyzed with monoclonal anti bodies, was immunopositive for measles hemagglutinin (HA) protein but not for measles nucleoprotein and rubella or mumps viral proteins. Thus the MMR antibody in autistic sera detected measles HA protein, which is unique to the measles subunit of the vaccine. Furthermore, over 90% of MMR antibody-positive autistic sera were also positive for MBP autoantibodies, suggesting a strong association between MMR and CNS autoimmunity in autism. Stemming from this evidence, we suggest that an inappropriate antibody response to MMR, specifically the measles component thereof, might be related to pathogenesis of autism.” http://www.ncbi.nlm.nih.gov/pubmed/12145534 Conclusions: Children vaccinated in infancy are at increased risk of hepatitis B virus infection in the late teens. The risk of chronic carriage after sexual exposure needs further assessment to determine if booster vaccines are necessary. http://www.bmj.com/content/325/7364/569 Abstract A traditional infectious disease vaccine is a preparation of live attenuated, inactivated or killed pathogen that stimulates immunity. Vaccine immunologic adjuvants are compounds incorporated into vaccines to enhance immunogenicity. Adjuvants have recently been implicated in the new syndrome named ASIA autoimmune/inflammatory syndrome induced by adjuvants. The objective describes the frequencies of post-vaccination clinical syndrome induced by adjuvants. We performed a cross-sectional study; adverse event following immunization was defined as any untoward medical occurrence that follows immunization 54 days prior to the event. Data on vaccinations and other risk factors were obtained from daily epidemiologic surveillance. Descriptive statistics were done using means and standard deviation, and odds ratio adjusted for potential confounding variables was calculated with SPSS 17 software. Forty-three out of 120 patients with moderate or severe manifestations following immunization were hospitalized from 2008 to 2011. All patients fulfilled at least 2 major and 1 minor criteria suggested by Shoenfeld and Agmon-Levin for ASIA diagnosis. The most frequent clinical findings were pyrexia 68 %, arthralgias 47 %, cutaneous disorders 33 %, muscle weakness 16 % and myalgias 14 %. Three patients had diagnosis of Guillain-Barre syndrome, one patient had Adult-Still’s disease 3 days after vaccination. A total of 76 % of the events occurred in the first 3 days post-vaccination. Two patients with previous autoimmune disease showed severe adverse reactions with the reactivation of their illness. Minor local reactions were present in 49 % of patients. Vaccines containing adjuvants may be associated with an increased risk of autoimmune/inflammatory adverse events following immunization http://www.ncbi.nlm.nih.gov/pubmed/23576057 Autoimmunity following hepatitis B vaccine as part of the spectrum of ‘Autoimmune (Auto-inflammatory) Syndrome induced by Adjuvants’ (ASIA): analysis of 93 cases. OBJECTIVES: In this study we analyzed the clinical and demographic manifestations among patients diagnosed with immune/autoimmune-mediated diseases post-hepatitis B vaccination. We aimed to find common denominators for all patients, regardless of different diagnosed diseases, as well as the correlation to the criteria of Autoimmune (Auto-inflammatory) Syndrome induced by Adjuvants (ASIA). PATIENTS AND METHODS:

“Unvaccinated children tended to be white, to have a mother who was married and had a college degree, to live in a household with an annual income exceeding $75,000 dollars, and to have parents who expressed concerns regarding the safety of vaccines and indicated that medical doctors have little influence over vaccination decisions for their children.” http://www.ncbi.nlm.nih.gov/pubmed/15231927 “Although persons often use vaccination and immunization interchangeably in reference to active immunization (VACCINES), the terms are not synonomous because the administration of an immunobiologic cannot be automatically equated with the development of adequate immunity.” http://www.cdc.gov/mmwr/PDF/rr/rr4301.pdf “Hib immunization contributed to an increased risk for H. influenzae type a meningitis through selection of  circulating H. influenzae type a clones. the incidence for H. influenzae type a meningitis increased 8-fold” http://jid.oxfordjournals.org/content/187/1/109.full.pdf+html “Virus-induced autoimmunity may play a causal role in autism. To examine the etiologic link of viruses in this brain disorder, we conducted a serologic study of measles virus, mumps virus, and rubella virus. Viral antibodies were measured by enzyme-linked immunosorbent assay in the serum of autistic children, normal children, and siblings of autistic children. The level of measles antibody, but not mumps or rubella antibodies, was significantly higher in autistic children as compared with normal children (P = 0.003) or siblings of autistic children (P <or= 0.0001). Furthermore, immunoblotting of measles vaccine virus revealed that the antibody was directed against a protein of approximately 74 kd molecular weight. The antibody to this antigen was found in 83% of autistic children but not in normal children or siblings of autistic children. Thus autistic children have a hyperimmune response to measles virus, which in the absence of a wild type of measles infection might be a sign of an abnormal immune reaction to the vaccine strain or virus reactivation.” http://www.ncbi.nlm.nih.gov/pubmed/12849883 “Our findings show a positive correlation between the number of vaccine doses administered and the percentage of hospitalizations and deaths. Since vaccines are given to millions of infants annually, it is imperative that health authorities have scientific data from synergistic toxicity studies on all combinations of vaccines that infants might receive. ” http://het.sagepub.com/content/31/10/1012.abstract?maxtoshow&HITS=10&hits=10&RESULTFORMAT&fulltext=vaccine+&andorexactfulltext=and&searchid=1&FIRSTINDEX=10&resourcetype=HWCIT Maternal transfer of mercury to the developing embryo/fetus: is there a safe level? “This study focused on standardized embryonic and fetal Hg exposures via primary exposure to the pregnant mother of two common Hg sources (dietary fish and parenteral vaccines). Data demonstrated that Hg exposures, particularly during the first trimester of pregnancy, at well-established dose/weight ratios produced severe damage to humans including death. ”  Toxicological & Environmental Chemistry Vol 94 2012 http://www.tandfonline.com/doi/full/10.1080/02772248.2012.724574 “reporting bias was too low to explain the magnitude increase in fetal-demise reporting rates in the VAERS database relative to the reported annual trends. Thus, a synergistic fetal toxicity likely resulted from the administration of both the pandemic (A-H1N1) and seasonal influenza vaccines during the 2009/2010 season.” http://www.ncbi.nlm.nih.gov/pubmed/23023030 “Hepatitis B vaccine might be followed by various rheumatic conditions and might trigger the onset of underlying inflammatory or autoimmune rheumatic diseases. ” http://www.ncbi.nlm.nih.gov/pubmed/10534549 “Autoimmunity to the central nervous system (CNS), especially to myelin basic protein (MBP), may play a causal role in autism, a neurodevelopmental disorder.